about
Recent developments in the detection and management of acute kidney injuryAdvances and unmet needs in genetic, basic and clinical science in Alport syndrome: report from the 2015 International Workshop on Alport SyndromeThe non-immunosuppressive management of childhood nephrotic syndromeGenetic Background is a Key Determinant of Glomerular Extracellular Matrix Composition and OrganizationNephrotic plasma alters slit diaphragm-dependent signaling and translocates nephrin, Podocin, and CD2 associated protein in cultured human podocytesHemopexin induces nephrin-dependent reorganization of the actin cytoskeleton in podocytes.Three-dimensional electron microscopy reveals the evolution of glomerular barrier injury.Tissue-specific host recognition by complement factor H is mediated by differential activities of its glycosaminoglycan-binding regions.Coinheritance of COL4A5 and MYO1E mutations accentuate the severity of kidney disease.Towards a biomarker panel for the assessment of AKI in children receiving intensive careER stress and basement membrane defects combine to cause glomerular and tubular renal disease resulting from Col4a1 mutations in mice.Epithelial-mesenchymal status influences how cells deposit fibrillin microfibrils.Intravenous methylprednisolone in idiopathic childhood nephrotic syndrome.Global analysis reveals the complexity of the human glomerular extracellular matrix.Glomerular cell cross-talk influences composition and assembly of extracellular matrixThe importance of podocyte adhesion for a healthy glomerulus.Applying Proteomics to Investigate Extracellular Matrix in Health and Disease.Be on alert for pediatric AKI.Glomerular cell crosstalk.Proteomic definitions of basement membrane composition in health and disease.Establishment of conditionally immortalized human glomerular mesangial cells in culture, with unique migratory properties.Saturated fatty acids induce insulin resistance in human podocytes: implications for diabetic nephropathy.The human glomerular podocyte is a novel target for insulin action.Alport Syndrome in Women and Girls.Glucocorticoid therapy regulates podocyte motility by inhibition of Rac1PLA2R binds to the annexin A2-S100A10 complex in human podocytes.Support for studies in paediatric medicine is needed.Identification of a major epitope recognized by PLA2R autoantibodies in primary membranous nephropathy.An inducible mouse model of podocin-mutation-related nephrotic syndrome.Vinculin is required to maintain glomerular barrier integrity.ACTB Loss-of-Function Mutations Result in a Pleiotropic Developmental Disorder.Genetic testing in steroid-resistant nephrotic syndrome: why, who, when and how?Prolonged exposure of mouse and human podocytes to insulin induces insulin resistance through lysosomal and proteasomal degradation of the insulin receptor.The Effects of Extensive Glomerular Filtration of Thin Graphene Oxide Sheets on Kidney Physiology.Basement Membrane Defects in Genetic Kidney Diseases.Should We Increase GFR with Bardoxolone in Alport Syndrome?Generation of Functioning Nephrons by Implanting Human Pluripotent Stem Cell-Derived Kidney Progenitors.Engineered basement membranes: from in vivo considerations to cell-based assaysPinpointing clinical diagnosis through whole exome sequencing to direct patient care: a case of Senior-Loken syndrome3D organoid-derived human glomeruli for personalised podocyte disease modelling and drug screening
P50
Q26738725-9EE7A110-00C6-4A8F-936A-0E4DD9944A58Q26748817-E30ABA73-AA24-408D-8DB5-6719485D5ED2Q26778157-C6C9BF3B-434F-4C35-BB92-1336CEF19383Q27342741-83959CB0-7E89-4911-89BF-767A6F5A9B41Q28303199-FAEDA369-1CCE-410B-A3D4-74E964365663Q30484244-F7218BB8-BA87-408C-9348-9A3D0F7EBD77Q30820232-BB335768-C9EC-4EC0-972A-79C8AE4D107FQ33405609-FB662C9C-0072-4217-8BD1-AB218BFE309CQ35952630-09C1AD8E-C94F-4DF5-90EC-F52EA7F29FC9Q35995907-A35FDEB8-9ADD-4AAD-B826-4551EF74F811Q36627721-8A4A260F-EA05-440C-9044-B19EB3E51B56Q37418467-61EC290F-5E2C-4E6D-827C-D0ADFDFFE772Q37682710-56A037F1-301E-4AF8-8796-9D9151E1D4D6Q37733558-4D38CB7D-AB9C-437B-8545-349C7F443017Q37733606-ACF3A1D0-65ED-4848-B45E-2E54831FA746Q38263509-400801BB-7BCB-43D3-AD8A-4BFFE4E297EDQ38649048-B3C5A08D-67F3-4E75-9B34-94554C993613Q38678506-F447EB44-134D-4FBA-8C26-6416D99E5292Q38793495-3D8D217B-1D81-4F28-8A63-9D24284BD3EDQ38935123-1E0335A5-C7A3-4510-B455-E1ED1BD9CA91Q39038233-EACA38CA-FC20-45F4-8778-26F1B1DCEA52Q39833131-93C87211-58E4-4E63-B28A-661DED3B074EQ40357961-EE3527B3-28C3-4ED1-B9A2-C216F26A3903Q41022440-5C0BDEFB-73B8-47F8-8A29-EB20A2F88FE3Q41159058-86C0726C-BFF7-42FE-B038-73782CE9D985Q41191481-C69257F6-2EC6-4D39-93C2-356B334299DCQ42705707-55853108-A50A-4352-A778-05EA7B803B01Q43123391-463C2987-B247-4B46-93D3-360C6E3E42F7Q43410167-FC61EB3A-2022-46EF-9542-68040ACA7B1CQ46507933-42218884-FAF7-4CDD-B706-6BE4D5EA95F5Q47315085-7E54FA65-9422-4429-8BE3-D04B0250AB79Q47357418-09DBADBA-5903-42A7-96E1-39773DAD2E33Q47885313-AE5E532C-DEBF-4E75-9769-A18909A99B58Q48168526-E79CED27-C94A-4DD6-A19D-43C24F9553DEQ49455564-091222C5-41FA-4CDB-BC77-E74246A4CC21Q49680628-60474751-5983-43DC-85CA-6D8BF3EE85A9Q50074638-240DE200-11A6-409D-AA4A-26AF6FEA6C76Q57181015-1AB38CF6-16A1-4780-9F3A-3B86287F48A3Q57206594-4D304DE5-A5A3-4410-A2CF-4AC46F05EE10Q59794197-531FF74D-58EC-422E-A65A-B9A5E8F9C688
P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Rachel Lennon
@ast
Rachel Lennon
@en
Rachel Lennon
@es
Rachel Lennon
@nl
Rachel Lennon
@sl
Рэйчел Леннон
@ru
type
label
Rachel Lennon
@ast
Rachel Lennon
@en
Rachel Lennon
@es
Rachel Lennon
@nl
Rachel Lennon
@sl
Рэйчел Леннон
@ru
prefLabel
Rachel Lennon
@ast
Rachel Lennon
@en
Rachel Lennon
@es
Rachel Lennon
@nl
Rachel Lennon
@sl
Рэйчел Леннон
@ru
P108
P106
P1153
9734493600
P21
P31
P496
0000-0001-6400-0227