about
Inducible arginase 1 deficiency in mice leads to hyperargininemia and altered amino acid metabolismStrategies to rescue the consequences of inducible arginase-1 deficiency in miceCardiac Hypertrophy Is Inhibited by a Local Pool of cAMP Regulated by Phosphodiesterase 2Novelty and SignificanceProof-of-Concept Gene Editing for the Murine Model of Inducible Arginase-1 Deficiencyβ-Arrestin 1 inhibits the GTPase-activating protein function of ARHGAP21, promoting activation of RhoA following angiotensin II type 1A receptor stimulation.Small heat shock protein 20 (Hsp20) facilitates nuclear import of protein kinase D 1 (PKD1) during cardiac hypertrophyLiver-specific knockout of arginase-1 leads to a profound phenotype similar to inducible whole body arginase-1 deficiency.Protein kinase D in the hypertrophy pathway.Arginase-1 deficiency.Heat shock protein 20 (HSP20) is a novel substrate for protein kinase D1 (PKD1).Specific interactions between Epac1, β-arrestin2 and PDE4D5 regulate β-adrenergic receptor subtype differential effects on cardiac hypertrophic signaling
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description
onderzoeker
@nl
researcher
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հետազոտող
@hy
name
Yuan Yan Sin
@ast
Yuan Yan Sin
@en
Yuan Yan Sin
@es
Yuan Yan Sin
@nl
Yuan Yan Sin
@sl
type
label
Yuan Yan Sin
@ast
Yuan Yan Sin
@en
Yuan Yan Sin
@es
Yuan Yan Sin
@nl
Yuan Yan Sin
@sl
prefLabel
Yuan Yan Sin
@ast
Yuan Yan Sin
@en
Yuan Yan Sin
@es
Yuan Yan Sin
@nl
Yuan Yan Sin
@sl
P106
P1153
37006043000
P31
P496
0000-0001-6537-5266