about
The oxysterol-binding protein homologue ORP1L interacts with Rab7 and alters functional properties of late endocytic compartmentsAn Overview of Helicobacter pylori VacA Toxin BiologyHelicobacter pylori Diversity and Gastric Cancer RiskThe host protein calprotectin modulates the Helicobacter pylori cag type IV secretion system via zinc sequestrationCrystal structure of the Helicobacter pylori vacuolating toxin p55 domainDynamic Computational Model of Symptomatic Bacteremia to Inform Bacterial Separation Treatment RequirementsAnalysis of surface-exposed outer membrane proteins in Helicobacter pylori.The vacuolating toxin from Helicobacter pylori forms hexameric pores in lipid bilayers at low pH.Mimicry of a host anion channel by a Helicobacter pylori pore-forming toxin.Helicobacter pylori and mitogen-activated protein kinases mediate activator protein-1 (AP-1) subcomponent protein expression and DNA-binding activity in gastric epithelial cells.Random mutagenesis of Helicobacter pylori vacA to identify amino acids essential for vacuolating cytotoxic activity.A 12-amino-acid segment, present in type s2 but not type s1 Helicobacter pylori VacA proteins, abolishes cytotoxin activity and alters membrane channel formationCarboxy-terminal proteolytic processing of Helicobacter pylori vacuolating toxin.Expression of Helicobacter pylori vacuolating toxin in Escherichia coli.Supporting data for analysis of the Helicobacter pylori exoproteomeHeterogeneity among Helicobacter pylori strains in expression of the outer membrane protein BabA.Genome sequence analysis of Helicobacter pylori strains associated with gastric ulceration and gastric cancer.Analysis of a beta-helical region in the p55 domain of Helicobacter pylori vacuolating toxin.In Helicobacter pylori auto-inducer-2, but not LuxS/MccAB catalysed reverse transsulphuration, regulates motility through modulation of flagellar gene transcriptionRegulation of gastric B cell recruitment is dependent on IL-17 receptor A signaling in a model of chronic bacterial infection.Analysis of protein expression regulated by the Helicobacter pylori ArsRS two-component signal transduction system.Genomic Comparison of cag pathogenicity island (PAI)-positive and -negative Helicobacter pylori strains: identification of novel markers for cag PAI-positive strainsIntercellular communication in Helicobacter pylori: luxS is essential for the production of an extracellular signaling moleculeHelicobacter pylori exploits a unique repertoire of type IV secretion system components for pilus assembly at the bacteria-host cell interfaceReconstitution of Helicobacter pylori VacA toxin from purified componentsGenes required for assembly of pili associated with the Helicobacter pylori cag type IV secretion systemEffective treatment of allergic airway inflammation with Helicobacter pylori immunomodulators requires BATF3-dependent dendritic cells and IL-10.Promoter analysis of Helicobacter pylori genes with enhanced expression at low pH.Molecular evolution of the Helicobacter pylori vacuolating toxin gene vacAInhibition of primary human T cell proliferation by Helicobacter pylori vacuolating toxin (VacA) is independent of VacA effects on IL-2 secretionGlobal analysis of Helicobacter pylori gene expression in human gastric mucosaIn search of the Helicobacter pylori VacA mechanism of action.Comparative genomic analysis of East Asian and non-Asian Helicobacter pylori strains identifies rapidly evolving genes.Functional plasticity in the type IV secretion system of Helicobacter pyloriProtein-protein interactions among Helicobacter pylori cag proteinsTwo different families of hopQ alleles in Helicobacter pylori.Colon-specific delivery of a probiotic-derived soluble protein ameliorates intestinal inflammation in mice through an EGFR-dependent mechanism.Role of connexin 43 in Helicobacter pylori VacA-induced cell death.Helicobacter pylori persistence: an overview of interactions between H. pylori and host immune defensesQuantitative effect of luxS gene inactivation on the fitness of Helicobacter pylori.
P50
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P1053
I-3814-2015
P106
P21
P31
P3829
P496
0000-0001-8503-002X
P569
2000-01-01T00:00:00Z