Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

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Altered ubiquitin-proteasome signaling in right ventricular hypertrophy and failure Bortezomib and heart failure: case-report and review of the French Pharmacovigilance database.Role of ubiquitin-proteasome system (UPS) in left ventricular hypertrophy (LVH).

P2860

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P2860

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

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http://www.wikidata.org/entity/Q42871160

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2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

2010年论文

@zh

2010年论文

@zh-cn

name

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@en

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@nl

type

Item

label

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@en

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@nl

prefLabel

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@en

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@nl

P1476

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

@en

P2093

Lucie Carrier

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P2860

Regulation of the stability and transcriptional activity of NFATc4 by ubiquitination

The ubiquitin-proteasome system and nonsense-mediated mRNA decay in hypertrophic cardiomyopathy

The ubiquitin-proteasome system in cardiac dysfunction

Proteasome functional insufficiency activates the calcineurin-NFAT pathway in cardiomyocytes and promotes maladaptive remodelling of stressed mouse hearts.

Calcineurin-NFAT signaling regulates the cardiac hypertrophic response in coordination with the MAPKs.

Proteasome inhibition decreases cardiac remodeling after initiation of pressure overload

Biochemical mechanisms of cardiac hypertrophy.

Molecular basis of bortezomib resistance: proteasome subunit beta5 (PSMB5) gene mutation and overexpression of PSMB5 protein.

Multiple cardiac proteasome subtypes differ in their susceptibility to proteasome inhibitors.

Bortezomib or high-dose dexamethasone for relapsed multiple myeloma.

P304

389-390

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P31

scholarly article

editorial

P356

10.1093/CVR/CVQ315

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2010-10-01T00:00:00Z

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P5875

47300282

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P698

20889761

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Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

about

P2860

P2860

Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698