about
The 2005 World Health Organization reevaluation of human and Mammalian toxic equivalency factors for dioxins and dioxin-like compoundsOsteopontin: a rapid and sensitive response to dioxin exposure in the osteoblastic cell line UMR-106A constitutively active dioxin/aryl hydrocarbon receptor induces stomach tumors.Health risk assessment procedures for endocrine disrupting compounds within different regulatory frameworks in the European Union.Risk to all or none? A comparative analysis of controversies in the health risk assessment of Bisphenol A.The basic helix-loop-helix-PAS protein ARNT functions as a potent coactivator of estrogen receptor-dependent transcriptionPhthalate diesters and their metabolites in human breast milk, blood or serum, and urine as biomarkers of exposure in vulnerable populations.Assessment factors--applications in health risk assessment of chemicals.Prenatal exposure to polychlorinated biphenyls (PCBs) and polybrominated diphenyl ethers (PBDEs) may influence birth weight among infants in a Swedish cohort with background exposure: a cross-sectional study.Model selection and evaluation for risk assessment of dioxin-contaminated sites.The influence of study design and sex-differences on results from developmental neurotoxicity studies of bisphenol A: implications for toxicity testing.Bridging the gap between academic research and regulatory health risk assessment of Endocrine Disrupting Chemicals.Exposure assessment at a PCDD/F contaminated site in Sweden--field measurements of exposure media and blood serum analysis.Toxicity of Bromkal 70-5DE, a technical mixture of polybrominated diphenyl ethers, following 28 d of oral exposure in rats and impact of analysed impurities.Transgenic mice with a constitutively active aryl hydrocarbon receptor display a gender-specific bone phenotype.Hepatic vitamin a depletion is a sensitive marker of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure in four rodent species.Facilitating the use of non-standard in vivo studies in health risk assessment of chemicals: a proposal to improve evaluation criteria and reporting.A constitutively active aryl hydrocarbon receptor causes loss of peritoneal B1 cells.Large variation in breast milk levels of organohalogenated compounds is dependent on mother's age, changes in body composition and exposures early in life.The dioxin/aryl hydrocarbon receptor mediates downregulation of osteopontin gene expression in a mouse model of gastric tumourigenesis.The constitutively active Ah receptor (CA-AhR) mouse as a model for dioxin exposure - effects in reproductive organs.The constitutively active Ah receptor (CA-Ahr) mouse as a potential model for dioxin exposure--effects in vital organs.Testing and refining the Science in Risk Assessment and Policy (SciRAP) web-based platform for evaluating the reliability and relevance of in vivo toxicity studiesScience in Risk Assessment and Policy (SciRAP): An Online Resource for Evaluating and ReportingIn Vivo(Eco)Toxicity StudiesGastric intestinal metaplasia in transgenic mice2,3,7,8-Tetrachlorodibenzo-p-dioxin affects retinol esterification in rat hepatic stellate cells and kidneyEffect of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on the hepatic stellate cell population in the rat
P50
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P50
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hulumtuese
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հետազոտող
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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Annika Hanberg
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P106
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P21
P31
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