Emerging role of IL-23/IL-17 axis in H pylori-associated pathology. - Wikidata - wikimedia - TriplyDB

Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

http://www.wikidata.org/entity/Q42911550

about

Comparative Immune Response in Children and Adults with H. pylori Infection Cytokines, cytokine gene polymorphisms and Helicobacter pylori infection: friend or foe?Helicobacter pylori vaccination: is there a path to protection?A MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.Fibroblastic colony-forming unit bone marrow cells delay progression to gastric dysplasia in a helicobacter model of gastric tumorigenesis Stat3: linking inflammation to epithelial cancer - more than a "gut" feeling?Genetic polymorphisms and tissue expression of interleukin-22 associated with risk and therapeutic response of gastric mucosa-associated lymphoid tissue lymphoma Stromal cells induce Th17 during Helicobacter pylori infection and in the gastric tumor microenvironment.Enterococcus faecalis infection causes inflammation, intracellular oxphos-independent ROS production, and DNA damage in human gastric cancer cells.IL-26 promotes the proliferation and survival of human gastric cancer cells by regulating the balance of STAT1 and STAT3 activation IFN-γ inhibits gastric carcinogenesis by inducing epithelial cell autophagy and T-cell apoptosis Activation of type I IFN signaling by NOD1 mediates mucosal host defense against Helicobacter pylori infection.Association of IL-17 polymorphisms with gastric cancer risk in Asian populations Defining the Roles of IFN-γ and IL-17A in Inflammation and Protection against Helicobacter pylori Infection.Role of interleukin-17 in chondrocytes of herniated intervertebral lumbar discs.Local Immune Response in Helicobacter pylori Infection Dendritic cells--why can they help and hurt us.Aspects of B-cell non-Hodgkin's lymphoma development: a transition from immune-reactivity to malignancy.Targeting IL-23 in human diseases.Role of virulence factors and host cell signaling in the recognition of Helicobacter pylori and the generation of immune responses.Targeting STAT3 in gastric cancer.STAT3: a critical component in the response to Helicobacter pylori infection.Increased expression of IL17A in human gastric cancer and its potential roles in gastric carcinogenesis.Alterations in Th17 and the Respective Cytokine Levels in Helicobacter pylori-Induced Stomach Diseases.The chemokine receptor CXCR5 is pivotal for ectopic mucosa-associated lymphoid tissue neogenesis in chronic Helicobacter pylori-induced inflammation.The association of interleukin-18 promoter polymorphisms and serum levels with duodenal ulcer, and their correlations with bacterial CagA and VacA virulence factors.Comparison of demographic characteristics and upper gastrointestinal endoscopy findings in HIV-positive, antiretroviral-treated patients with and without Helicobacter pylori coinfection.Associations of Functional MicroRNA Binding Site Polymorphisms in IL23/Th17 Inflammatory Pathway Genes with Gastric Cancer Risk.Association of the CagA status of Helicobacter pylori and serum levels of interleukin (IL)-17 and IL-23 in duodenal ulcer patients.Stromal C-type lectin receptor COLEC12 integrates H. pylori, PGE2-EP2/4 axis and innate immunity in gastric diseases.The presence of Helicobacter pylori in the liver depends on the Th1, Th17 and Treg cytokine profile of the patient.Association between polymorphisms in interleukin-17A and interleukin-17F genes and risks of gastric cancer.Analysis of the association of interleukin-17 gene polymorphisms with gastric cancer risk and interaction with Helicobacter pylori infection in a Chinese population.Interleukin-17A Promotes Parietal Cell Atrophy by Inducing Apoptosis.IL-17A and IL-17F Gene Expression is Strongly Induced in the Mucosa of H. pylori-Infected Subjects From Kenya and Germany

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Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

http://www.wikidata.org/entity/Q42911550

description

2007 nî lūn-bûn

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name

Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

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Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

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type

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Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

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Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

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prefLabel

Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

@en

Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

@nl

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WJG.V13.I42.5547

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World Journal of Gastroenterology

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Emerging role of IL-23/IL-17 axis in H pylori-associated pathology.

@en

P2093

Francesco Pallone

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Giovanni Monteleone

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Roberta Caruso

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P2860

Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells

Molecular characterization of the 128-kDa immunodominant antigen of Helicobacter pylori associated with cytotoxicity and duodenal ulcer

Inflammation, atrophy, and gastric cancer

Interleukin-23 drives innate and T cell-mediated intestinal inflammation

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Matrix metalloproteinases

STAT3 and NF-kappaB signal pathway is required for IL-23-mediated IL-17 production in spontaneous arthritis animal model IL-1 receptor antagonist-deficient mice

Transforming growth factor-beta induces development of the T(H)17 lineage

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5547-5551

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10.3748/WJG.V13.I42.5547

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17948927

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Helicobacter pylori

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4172732

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