Cre-loxP DNA recombination is possible with only minimal unspecific transcriptional changes and without cardiomyopathy in Tg(alphaMHC-MerCreMer) mice.
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A Murine Myh6MerCreMer Knock-In Allele Specifically Mediates Temporal Genetic Deletion in Cardiomyocytes after Tamoxifen InductionDirect, differential effects of tamoxifen, 4-hydroxytamoxifen, and raloxifene on cardiac myocyte contractility and calcium handlingGeneration of a tamoxifen inducible Tnnt2MerCreMer knock-in mouse model for cardiac studiesSustained Toll-Like Receptor 9 Activation Promotes Systemic and Cardiac Inflammation, and Aggravates Diastolic Heart Failure in SERCA2a KO MiceMutation of the calmodulin binding motif IQ of the L-type Ca(v)1.2 Ca2+ channel to EQ induces dilated cardiomyopathy and death.Ca(2+)-Clock-Dependent Pacemaking in the Sinus Node Is Impaired in Mice with a Cardiac Specific Reduction in SERCA2 AbundanceHeme oxygenase-1 expression protects the heart from acute injury caused by inducible Cre recombinase.Moderate and high amounts of tamoxifen in αMHC-MerCreMer mice induce a DNA damage response, leading to heart failure and death.Cardiac fibrosis in mice expressing an inducible myocardial-specific Cre driver.SERCA2 activity is involved in the CNP-mediated functional responses in failing rat myocardium.Alterations in sarcomere function modify the hyperplastic to hypertrophic transition phase of mammalian cardiomyocyte development.Chromosomal mapping of the αMHC-MerCreMer transgene in mice reveals a large genomic deletion.The effect of raloxifene on left ventricular hypertrophy in postmenopausal women: A prospective, randomized, and controlled study.Impaired left ventricular mechanical and energetic function in mice after cardiomyocyte-specific excision of Serca2.PI3Kα is essential for the recovery from Cre/tamoxifen cardiotoxicity and in myocardial insulin signalling but is not required for normal myocardial contractility in the adult heart.
P2860
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P2860
Cre-loxP DNA recombination is possible with only minimal unspecific transcriptional changes and without cardiomyopathy in Tg(alphaMHC-MerCreMer) mice.
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2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Cre-loxP DNA recombination is ...... d without cardiomyopathy in Tg
@nl
Cre-loxP DNA recombination is ...... n Tg(alphaMHC-MerCreMer) mice.
@en
type
label
Cre-loxP DNA recombination is ...... d without cardiomyopathy in Tg
@nl
Cre-loxP DNA recombination is ...... n Tg(alphaMHC-MerCreMer) mice.
@en
prefLabel
Cre-loxP DNA recombination is ...... d without cardiomyopathy in Tg
@nl
Cre-loxP DNA recombination is ...... n Tg(alphaMHC-MerCreMer) mice.
@en
P2093
P2860
P50
P1476
Cre-loxP DNA recombination is ...... n Tg(alphaMHC-MerCreMer) mice.
@en
P2093
Karina Hougen
Kristin B Andersson
Mathis K Stokke
Stale Nygard
Ulla Enger
P2860
P304
P356
10.1152/AJPHEART.01155.2009
P577
2010-08-27T00:00:00Z