Green tea: a new option for the prevention or control of osteoarthritis.

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2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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name

Green tea: a new option for the prevention or control of osteoarthritis.

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Green tea: a new option for the prevention or control of osteoarthritis.

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Green tea: a new option for the prevention or control of osteoarthritis.

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Green tea: a new option for the prevention or control of osteoarthritis.

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prefLabel

Green tea: a new option for the prevention or control of osteoarthritis.

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Green tea: a new option for the prevention or control of osteoarthritis.

@nl

P1476

Green tea: a new option for the prevention or control of osteoarthritis.

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P2093

Chander Raman

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Santosh K Katiyar

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P2860

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

Green tea polyphenols prevent UV-induced immunosuppression by rapid repair of DNA damage and enhancement of nucleotide excision repair genes

The encephalitogenicity of T(H)17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF.

Prevention of collagen-induced arthritis in mice by a polyphenolic fraction from green tea.

Epigallocatechin-3-gallate suppresses the global interleukin-1beta-induced inflammatory response in human chondrocytes.

Inhibition of UVB-induced skin tumor development by drinking green tea polyphenols is mediated through DNA repair and subsequent inhibition of inflammation.

Large-scale meta-analysis of interleukin-1 beta and interleukin-1 receptor antagonist polymorphisms on risk of radiographic hip and knee osteoarthritis and severity of knee osteoarthritis.

Epigallocatechin-3-gallate inhibits interleukin-1beta-induced expression of nitric oxide synthase and production of nitric oxide in human chondrocytes: suppression of nuclear factor kappaB activation by degradation of the inhibitor of nuclear factor

Green tea polyphenol epigallocatechin-3-gallate inhibits the IL-1 beta-induced activity and expression of cyclooxygenase-2 and nitric oxide synthase-2 in human chondrocytes.

Gene deletion of either interleukin-1beta, interleukin-1beta-converting enzyme, inducible nitric oxide synthase, or stromelysin 1 accelerates the development of knee osteoarthritis in mice after surgical transection of the medial collateral ligament

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2011-08-10T00:00:00Z

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osteoarthritis

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