Frequencies of HCV-specific effector CD4+ T cells by flow cytometry: correlation with clinical disease stages. - Wikidata - wikimedia - TriplyDB

Frequencies of HCV-specific effector CD4+ T cells by flow cytometry: correlation with clinical disease stages.

Frequencies of HCV-specific effector CD4+ T cells by flow cytometry: correlation with clinical disease stages.

http://www.wikidata.org/entity/Q43036009

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Hepatitis C virus infection in the immunocompromised host: a complex scenario with variable clinical impact Acute hepatitis C: prevention and treatment Lymphocyte subsets in hemophilic patients with hepatitis C virus infection with or without human immunodeficiency virus co-infection: a nested cross-sectional study Cellular immune response to hepatitis-C-virus in subjects without viremia or seroconversion: is it important?Effect of bottlenecking on evolution of the nonstructural protein 3 gene of hepatitis C virus during sexually transmitted acute resolving infection Hepatitis C virus core protein leads to immune suppression and liver damage in a transgenic murine model Broad Specificity of Virus-Specific CD4+ T-Helper-Cell Responses in Resolved Hepatitis C Virus Infection Evaluating Replication-Defective Vesicular Stomatitis Virus as a Vaccine Vehicle Immunization with hepatitis C virus-like particles protects mice from recombinant hepatitis C virus-vaccinia infection.Ex vivo analysis of human memory CD4 T cells specific for hepatitis C virus using MHC class II tetramers Acute hepatitis C virus infection: A chronic problem Hepatitis C Virus-Specific T-Cell Gamma Interferon and Proliferative Responses Are More Common in Perihepatic Lymph Nodes than in Peripheral Blood or Liver Factors influencing a low rate of hepatitis C viral RNA clearance in heroin users from Southern China Female sex and IL28B, a synergism for spontaneous viral clearance in hepatitis C virus (HCV) seroconverters from a community-based cohort Adaptive immune responses in acute and chronic hepatitis C virus infection Statistical linkage analysis of substitutions in patient-derived sequences of genotype 1a hepatitis C virus nonstructural protein 3 exposes targets for immunogen design Immunotherapy for HCV infection: next steps.Frequency of gC1qR+CD4+ T cells increases during acute hepatitis C virus infection and remains elevated in patients with chronic infection.Genetic immunization and comprehensive screening approaches in HLA-A2 transgenic mice lead to the identification of three novel epitopes in hepatitis C virus NS3 antigen.Antigen-specific immune responses and liver histology in HIV and hepatitis C coinfection.Characterization of the specific CD4+ T cell response against the F protein during chronic hepatitis C virus infection The protective role of myeloid-derived suppressor cells in concanavalin A-induced hepatic injury Immunization with hepatitis C virus-like particles induces humoral and cellular immune responses in nonhuman primates.Control of heterologous hepatitis C virus infection in chimpanzees is associated with the quality of vaccine-induced peripheral T-helper immune response Two distinct functional patterns of hepatitis C Virus (HCV)-specific T cell responses in seronegative, aviremic patients Strong hepatitis C virus (HCV)-specific cell-mediated immune responses in the absence of viremia or antibodies among uninfected siblings of HCV chronically infected children.Opposing roles of STAT1 and STAT3 in T cell-mediated hepatitis: regulation by SOCS.Pathogenesis of hepatitis C virus recurrence in the liver allograft.Characterization of antigenic variants of hepatitis C virus in immune evasion.Molecular and contextual markers of hepatitis C virus and drug abuse.Broadly directed virus-specific CD4+ T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence.Hepatitis C virus-multispecific T-cell responses without viremia or seroconversion among Egyptian health care workers at high risk of infection.Cytokines and HCV-related disorders.Ex vivo monitoring of antigen-specific CD4+ T cells after recall immunization with tetanus toxoid.Development of prophylactic and therapeutic vaccines against hepatitis C virus.DNA vaccination protects mice against challenge with Listeria monocytogenes expressing the hepatitis C virus NS3 protein.Spontaneous recovery in acute human hepatitis C virus infection: functional T-cell thresholds and relative importance of CD4 help.Host and viral factors in the immunopathogenesis of primary hepatitis C virus infection.TH1 cytokine response to HCV peptides in Egyptian health care workers: a pilot study.Functionality of the immune system in patients with chronic hepatitis C: trial by superinfections and vaccinations.

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Frequencies of HCV-specific effector CD4+ T cells by flow cytometry: correlation with clinical disease stages.

http://www.wikidata.org/entity/Q43036009

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Frequencies of HCV-specific ef ...... with clinical disease stages.

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Andrew J Conrad

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Anna W Sasaki

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Antony Bakke

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Camette Miner

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David J Hinrichs

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H G Archie Bouwer

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Hugo R Rosen

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P2860

Viral immune evasion due to persistence of activated T cells without effector function

Hepatitis C virus (HCV) specific immune responses in anti-HCV positive patients without hepatitis C viraemia

Compartmentalization of T lymphocytes to the site of disease: intrahepatic CD4+ T cells specific for the protein NS4 of hepatitis C virus in patients with chronic hepatitis C

Classification of hepatitis C virus into six major genotypes and a series of subtypes by phylogenetic analysis of the NS-5 region

Vigorous HIV-1-specific CD4+ T cell responses associated with control of viremia

Analysis of a successful immune response against hepatitis C virus

Global surveillance and control of hepatitis C. Report of a WHO Consultation organized in collaboration with the Viral Hepatitis Prevention Board, Antwerp, Belgium

Possible mechanism involving T-lymphocyte response to non-structural protein 3 in viral clearance in acute hepatitis C virus infection

Cellular immune responses persist and humoral responses decrease two decades after recovery from a single-source outbreak of hepatitis C

Replication of hepatitis C virus

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