ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
about
High fluorescence in situ hybridization percentage of deletion 11q in patients with chronic lymphocytic leukemia is an independent predictor of adverse outcomeMolecular pathogenesis of chronic lymphocytic leukemia.Gene expression signatures but not cell cycle checkpoint functions distinguish AT carriers from normal individualsMolecular pathogenesis of CLL and its evolution.The cell cycle checkpoint inhibitors in the treatment of leukemias.Clinicopathological significance of ATM-Chk2 expression in sporadic breast cancers: a comprehensive analysis in large cohorts.Identification of molecular and functional patterns of p53 alterations in chronic lymphocytic leukemia patients in different phases of the diseaseCLL/SLL diagnosed in an adolescent.Telomere shortening, TP53 mutations and deletions in chronic lymphocytic leukemia result in increased chromosomal instability and breakpoint clustering in heterochromatic regions.
P2860
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P2860
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@en
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@nl
type
label
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@en
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@nl
prefLabel
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@en
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@nl
P2860
P1433
P1476
ATM and chronic lymphocytic leukemia: mutations, and not only deletions, matter.
@en
P2093
Davide Rossi
P2860
P356
10.3324/HAEMATOL.2011.057109
P577
2012-01-01T00:00:00Z