about
In vivo molecular imaging of experimental joint inflammation by combined (18)F-FDG positron emission tomography and computed tomography.Dominant recognition of a Borrelia burgdorferi outer surface protein A peptide by T helper cells in patients with treatment-resistant Lyme arthritisThe role and clinical implications of G6PI in experimental models of rheumatoid arthritisLyme disease and current aspects of immunization.Lack of IL-4 receptor expression on T helper cells reduces T helper 2 cell polyfunctionality and confers resistance in allergic bronchopulmonary mycosisParacrine IL-33 stimulation enhances lipopolysaccharide-mediated macrophage activationB cell depletion reduces the number of autoreactive T helper cells and prevents glucose-6-phosphate isomerase-induced arthritisAssessment of unspecific near-infrared dyes in laser-induced fluorescence imaging of experimental arthritis.Immunization with an immunodominant self-peptide derived from glucose-6-phosphate isomerase induces arthritis in DBA/1 mice.Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.Multivariate analysis of flow cytometric data using decision treesTim-3 inhibits T helper type 1-mediated auto- and alloimmune responses and promotes immunological tolerance.DRB1*0401-restricted human T cell clone specific for the major proinsulin73-90 epitope expresses a down-regulatory T helper 2 phenotype.Self-tolerance in the immune privileged CNS: lessons from the entorhinal cortex lesion model.Towards the generation of B-cell receptor retrogenic miceSubthreshold IKK activation modulates the effector functions of primary mast cells and allows specific targeting of transformed mast cells.Simultaneous inhibition of JAK and SYK kinases ameliorates chronic and destructive arthritis in mice.The T helper cell response in Lyme arthritis: differential recognition of Borrelia burgdorferi outer surface protein A in patients with treatment-resistant or treatment-responsive Lyme arthritisTAK1 and IKK2, novel mediators of SCF-induced signaling and potential targets for c-Kit-driven diseases.Increased frequency of EBV-specific effector memory CD8+ T cells correlates with higher viral load in rheumatoid arthritis.Tim-3 is differently expressed in genetically susceptible C57BL/6 and resistant BALB/c mice during oral infection with Toxoplasma gondii.IL-10 mediates plasmacytosis-associated immunodeficiency by inhibiting complement-mediated neutrophil migration.Immunometabolism and autoimmunity.Histamine modulates γδ-T lymphocyte migration and cytotoxicity, via Gi and Gs protein-coupled signalling pathways.Lipopeptides of Borrelia burgdorferi outer surface proteins induce Th1 phenotype development in alphabeta T-cell receptor transgenic mice.A Th17-like developmental process leads to CD8(+) Tc17 cells with reduced cytotoxic activity.Lysophosphatidic acid inhibits the cytotoxic activity of NK cells: involvement of Gs protein-mediated signaling.In vivo activation of Treg cells with a CD28 superagonist prevents and ameliorates chronic destructive arthritis in mice.Were Antibody Titers the Right Parameter When Immunologically Comparing Pneumococcal Vaccines?Death ligand TRAIL induces no apoptosis but inhibits activation of human (auto)antigen-specific T cells.Potential infectious agents in the induction of arthritides.Pertussis toxin prevents the induction of peripheral T cell anergy and enhances the T cell response to an encephalitogenic peptide of myelin basic protein.18 F-Fluoride positron emission tomography/computed tomography for noninvasive in vivo quantification of pathophysiological bone metabolism in experimental murine arthritis.Diversity and flexibility of Th17 effector functions.Amelioration of experimental arthritis by stroke-induced immunosuppression is independent of Treg cell function.IL-17A secretion by CD8+ T cells supports Th17-mediated autoimmune encephalomyelitis.Crucial role of the interleukin 1 receptor family member T1/ST2 in T helper cell type 2-mediated lung mucosal immune responses.Regulatory CD4+CD25+ T cells restrict memory CD8+ T cell responses.Autoantibodies against glucose-6-phosphate isomerase are not a diagnostic marker for juvenile idiopathic arthritis.Inducible costimulator (ICOS) blockade inhibits accumulation of polyfunctional T helper 1/T helper 17 cells and mitigates autoimmune arthritis.
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description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
@hy
name
Thomas Kamradt
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Thomas Kamradt
@en
Thomas Kamradt
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Thomas Kamradt
@nl
Thomas Kamradt
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type
label
Thomas Kamradt
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Thomas Kamradt
@en
Thomas Kamradt
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Thomas Kamradt
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Thomas Kamradt
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prefLabel
Thomas Kamradt
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Thomas Kamradt
@en
Thomas Kamradt
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Thomas Kamradt
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Thomas Kamradt
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P106
P108
P1153
20934555500
P21
P31
P496
0000-0001-8443-5893