Oligodendrocytes in mouse corpus callosum are coupled via gap junction channels formed by connexin47 and connexin32.
about
Gap junctions in inherited human disorders of the central nervous system.Myelinating satellite oligodendrocytes are integrated in a glial syncytium constraining neuronal high-frequency activityEffect of Lead (Pb) on Inflammatory Processes in the BrainClassic 18.5- and 21.5-kDa myelin basic protein isoforms associate with cytoskeletal and SH3-domain proteins in the immortalized N19-oligodendroglial cell line stimulated by phorbol ester and IGF-1.Cx32 and Cx47 mediate oligodendrocyte:astrocyte and oligodendrocyte:oligodendrocyte gap junction coupling.Connexin26 expression in brain parenchymal cells demonstrated by targeted connexin ablation in transgenic mice.Beyond cell replacement: unresolved roles of NG2-expressing progenitorsActivated microglia do not form functional gap junctions in vivo.Pathologic and phenotypic alterations in a mouse expressing a connexin47 missense mutation that causes Pelizaeus-Merzbacher-like disease in humans.The role of gap junction channels during physiologic and pathologic conditions of the human central nervous systemDeletion of oligodendrocyte Cx32 and astrocyte Cx43 causes white matter vacuolation, astrocyte loss and early mortality.A new mutation in GJC2 associated with subclinical leukodystrophy.Germ-line recombination activity of the widely used hGFAP-Cre and nestin-Cre transgenes.Glial Regulation of the Neuronal Connectome through Local and Long-Distant Communication.Gap junction communication in myelinating gliaVoltage-dependent K+ currents contribute to heterogeneity of olfactory ensheathing cellsCharacterization of Panglial Gap Junction Networks in the Thalamus, Neocortex, and Hippocampus Reveals a Unique Population of Glial Cells.Activated immune response in an inherited leukodystrophy disease caused by the loss of oligodendrocyte gap junctions.Brain connexins in demyelinating diseases: therapeutic potential of glial targetsEstimating functional connectivity in an electrically coupled interneuron network.Loss of Coupling Distinguishes GJB1 Mutations Associated with CNS Manifestations of CMT1X from Those Without CNS Manifestations.Synapses between NG2 glia and neurons.Astrocyte dysfunction in temporal lobe epilepsy: K+ channels and gap junction coupling.Connexins-mediated glia networking impacts myelination and remyelination in the central nervous system.Mix and match: investigating heteromeric and heterotypic gap junction channels in model systems and native tissues.GJB1-associated X-linked Charcot-Marie-Tooth disease, a disorder affecting the central and peripheral nervous systems.Oligodendroglia: metabolic supporters of neurons.CD38 positively regulates postnatal development of astrocytes cell-autonomously and oligodendrocytes non-cell-autonomously.Glioma-associated microglia and macrophages/monocytes display distinct electrophysiological properties and do not communicate via gap junctionsConnexins and pannexins: At the junction of neuro-glial homeostasis & disease.Damage to myelin and oligodendrocytes: a role in chronic outcomes following traumatic brain injury?Oligodendroglial excitability mediated by glutamatergic inputs and Nav1.2 activation.Increased Cx32 expression in spinal cord TrkB oligodendrocytes following peripheral axon injury.Functional heterotypic interactions between astrocyte and oligodendrocyte connexins.The distribution and functional properties of Pelizaeus-Merzbacher-like disease-linked Cx47 mutations on Cx47/Cx47 homotypic and Cx47/Cx43 heterotypic gap junctions.Antisense oligodeoxynucleotides targeting connexin43 reduce cerebral astrocytosis and edema in a rat model of traumatic brain injury.Loss of astrocyte connexins 43 and 30 does not significantly alter susceptibility or severity of acute experimental autoimmune encephalomyelitis in mice.Astrocyte Sodium Signalling and Panglial Spread of Sodium Signals in Brain White Matter.Epilepsy and astrocyte energy metabolism.Pelizaeus-Merzbacher-Like Disease in a Family With Variable Phenotype and a Novel Splicing GJC2 Mutation.
P2860
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P2860
Oligodendrocytes in mouse corpus callosum are coupled via gap junction channels formed by connexin47 and connexin32.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@en
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@nl
type
label
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@en
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@nl
prefLabel
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@en
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@nl
P2093
P356
P1433
P1476
Oligodendrocytes in mouse corp ...... by connexin47 and connexin32.
@en
P2093
Brigitte Haas
Helmut Kettenmann
Khalad Karram
Klaus Willecke
Marta Maglione
Oliver Tress
P304
P356
10.1002/GLIA.20991
P577
2010-07-01T00:00:00Z