Electrophysiological abnormalities in both axotomized and nonaxotomized pyramidal neurons following mild traumatic brain injury.
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Microglia processes associate with diffusely injured axons following mild traumatic brain injury in the micro pig.Recovery of neurological function despite immediate sleep disruption following diffuse brain injury in the mouse: clinical relevance to medically untreated concussionMild Traumatic Brain Injury Evokes Pyramidal Neuron Axon Initial Segment Plasticity and Diffuse Presynaptic Inhibitory Terminal Loss.A Mechanistic End-to-End Concussion Model That Translates Head Kinematics to Neurologic Injury.Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.Stimulation-induced ectopicity and propagation windows in model damaged axons.Moderately elevated intracranial pressure after diffuse traumatic brain injury is associated with exacerbated neuronal pathology and behavioral morbidity in the rat.Neuroimaging biomarkers in mild traumatic brain injury (mTBI).Computational Models Describing Possible Mechanisms for Generation of Excessive Beta Oscillations in Parkinson's Disease.Increased Network Excitability Due to Altered Synaptic Inputs to Neocortical Layer V Intact and Axotomized Pyramidal Neurons after Mild Traumatic Brain InjuryTherapy development for diffuse axonal injury.The p38α MAPK regulates microglial responsiveness to diffuse traumatic brain injury.Mild traumatic brain injury in the mouse induces axotomy primarily within the axon initial segmentKnockout of Cyclophilin-D Provides Partial Amelioration of Intrinsic and Synaptic Properties Altered by Mild Traumatic Brain Injury.Diffuse traumatic axonal injury in the optic nerve does not elicit retinal ganglion cell loss.Scale and pattern of atrophy in the chronic stages of moderate-severe TBI.Cytokine cascades induced by mechanical trauma injury alter voltage-gated sodium channel activity in intact cortical neurons.Control of Retinal Ganglion Cell Positioning and Neurite Growth: Combining 3D Printing with Radial Electrospun Scaffolds.Mild Traumatic Brain Injury Induces Structural and Functional Disconnection of Local Neocortical Inhibitory Networks via Parvalbumin Interneuron Diffuse Axonal Injury.Pathophysiology and Treatment of Memory Dysfunction After Traumatic Brain Injury.Distal axotomy enhances retrograde presynaptic excitability onto injured pyramidal neurons via trans-synaptic signaling.Compromised axon initial segment integrity in EAE is preceded by microglial reactivity and contact.Multi-scale, multi-modal analysis uncovers complex relationship at the brain tissue-implant neural interface: New Emphasis on the Biological Interface.Gabapentin Prevents Progressive Increases in Excitatory Connectivity and Epileptogenesis Following Neocortical Trauma.
P2860
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P2860
Electrophysiological abnormalities in both axotomized and nonaxotomized pyramidal neurons following mild traumatic brain injury.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Electrophysiological abnormali ...... g mild traumatic brain injury.
@en
Electrophysiological abnormali ...... g mild traumatic brain injury.
@nl
type
label
Electrophysiological abnormali ...... g mild traumatic brain injury.
@en
Electrophysiological abnormali ...... g mild traumatic brain injury.
@nl
prefLabel
Electrophysiological abnormali ...... g mild traumatic brain injury.
@en
Electrophysiological abnormali ...... g mild traumatic brain injury.
@nl
P2093
P2860
P1476
Electrophysiological abnormali ...... g mild traumatic brain injury.
@en
P2093
John E Greer
John T Povlishock
Kimberle M Jacobs
P2860
P304
P356
10.1523/JNEUROSCI.0881-12.2012
P407
P577
2012-05-01T00:00:00Z