Upregulation of AT1R and iNOS in the rostral ventrolateral medulla (RVLM) is essential for the sympathetic hyperactivity and hypertension in the 2K-1C Wistar rat model.
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All's well that transcribes well: non-coding RNAs and post-stroke brain damageMicroRNA in ischemic stroke etiology and pathology.Differential activation of adrenal, renal, and lumbar sympathetic nerves following stimulation of the rostral ventrolateral medulla of the ratAltered regulation of the rostral ventrolateral medulla in hypertensive obese Zucker rats.Melatonin attenuates hypertension-induced renal injury partially through inhibiting oxidative stress in rats.Sympathetic overactivity occurs before hypertension in the two-kidney, one-clip model.Hypertension in mice with transgenic activation of the brain renin-angiotensin system is vasopressin dependentAcupuncture effect and central autonomic regulation.Swimming Training Modulates Nitric Oxide-Glutamate Interaction in the Rostral Ventrolateral Medulla in Normotensive Conscious RatsNeural Control of Non-vasomotor Organs in Hypertension.Oxidative stress in the cardiovascular center has a pivotal role in the sympathetic activation in hypertension.The crosstalk between the kidney and the central nervous system: the role of renal nerves in blood pressure regulation.Effects of mesenchymal stem cells in renovascular hypertension.Mechanisms of renal sympathetic activation in renovascular hypertension.Mesenchymal stem cells and chronic renal artery stenosis.Compromised blood-brain barrier permeability: novel mechanism by which circulating angiotensin II signals to sympathoexcitatory centres during hypertension.Exercise training restores oxidative stress and nitric oxide synthases in the rostral ventrolateral medulla of renovascular hypertensive rats.Oxidative stress in the brain causes hypertension via sympathoexcitation.Effects of salt loading on sympathetic activity and blood pressure in anesthetized two-kidney, one clip hypertensive rats.Losartan reduces oxidative stress within the rostral ventrolateral medulla of rats with renovascular hypertension.Modulation of inducible nitric oxide synthase (iNOS) expression and cardiovascular responses during static exercise following iNOS antagonism within the ventrolateral medulla.Electroacupuncture and moxibustion decrease renal sympathetic nerve activity and retard progression of renal disease in rats.Aldosterone Contributes to Sympathoexcitation in Renovascular Hypertension.Increased Dietary Salt Changes Baroreceptor Sensitivity and Intrarenal Renin-Angiotensin System in Goldblatt Hypertension.
P2860
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P2860
Upregulation of AT1R and iNOS in the rostral ventrolateral medulla (RVLM) is essential for the sympathetic hyperactivity and hypertension in the 2K-1C Wistar rat model.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Upregulation of AT1R and iNOS ...... in the 2K-1C Wistar rat model.
@en
Upregulation of AT1R and iNOS in the rostral ventrolateral medulla
@nl
type
label
Upregulation of AT1R and iNOS ...... in the 2K-1C Wistar rat model.
@en
Upregulation of AT1R and iNOS in the rostral ventrolateral medulla
@nl
prefLabel
Upregulation of AT1R and iNOS ...... in the 2K-1C Wistar rat model.
@en
Upregulation of AT1R and iNOS in the rostral ventrolateral medulla
@nl
P2093
P2860
P50
P356
P1476
Upregulation of AT1R and iNOS ...... in the 2K-1C Wistar rat model
@en
P2093
Elizabeth Barbosa de Oliveira-Sales
Miriam Sterman Dolnikoff
Mirian Aparecida Boim
P2860
P304
P356
10.1038/AJH.2010.64
P577
2010-04-01T00:00:00Z