Increase of GABAA receptor-mediated tonic inhibition in dentate granule cells after traumatic brain injury.
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Making Waves in the Brain: What Are Oscillations, and Why Modulating Them Makes Sense for Brain InjuryA stress steroid triggers anxiety via increased expression of α4βδ GABAA receptors in methamphetamine dependence.Synaptic reorganization of inhibitory hilar interneuron circuitry after traumatic brain injury in miceTraumatic Brain Injury Increases Cortical Glutamate Network Activity by Compromising GABAergic ControlKnockout of the γ-aminobutyric acid receptor subunit α4 reduces functional δ-containing extrasynaptic receptors in hippocampal pyramidal cells at the onset of puberty.Diabetes induces GABA receptor plasticity in murine vagal motor neurons.Regulation of the surface expression of α4β2δ GABAA receptors by high efficacy statesGABA(A) receptor regulation after experimental traumatic brain injury.Decoding hippocampal signaling deficits after traumatic brain injuryα4βδ GABAA receptors and tonic inhibitory current during adolescence: effects on mood and synaptic plasticityNeurosteroid interactions with synaptic and extrasynaptic GABA(A) receptors: regulation of subunit plasticity, phasic and tonic inhibition, and neuronal network excitability.Neuropathophysiology of Brain Injury.Neural circuit mechanisms of post-traumatic epilepsy.Biomarkers of cognitive dysfunction in traumatic brain injury.GABA receptors in brain development, function, and injury.Anticonvulsive evaluation of THIP in the murine pentylenetetrazole kindling model: lack of anticonvulsive effect of THIP despite functional δ-subunit-containing GABAA receptors in dentate gyrus granule cells.Differential expression of microRNAs in the brains of mice subjected to increasing grade of mild traumatic brain injury.Pathophysiology and Treatment of Memory Dysfunction After Traumatic Brain Injury.Septohippocampal Neuromodulation Improves Cognition after Traumatic Brain Injury.Differential effects of rapamycin treatment on tonic and phasic GABAergic inhibition in dentate granule cells after focal brain injury in mice.GABA-independent GABAA receptor openings maintain tonic currents.How and Why Study Posttraumatic Epileptogenesis in Animal Models?Spontaneous recovery of traumatic brain injury-induced functional deficits is not hindered by daily administration of lorazepam.The GABAA antagonist DPP-4-PIOL selectively antagonises tonic over phasic GABAergic currents in dentate gyrus granule cells.Neurosteroid regulation of GABA receptors: A role in catamenial epilepsy
P2860
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P2860
Increase of GABAA receptor-mediated tonic inhibition in dentate granule cells after traumatic brain injury.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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name
Increase of GABAA receptor-med ...... after traumatic brain injury.
@en
Increase of GABAA receptor-med ...... after traumatic brain injury.
@nl
type
label
Increase of GABAA receptor-med ...... after traumatic brain injury.
@en
Increase of GABAA receptor-med ...... after traumatic brain injury.
@nl
prefLabel
Increase of GABAA receptor-med ...... after traumatic brain injury.
@en
Increase of GABAA receptor-med ...... after traumatic brain injury.
@nl
P2093
P1476
Increase of GABAA receptor-med ...... after traumatic brain injury.
@en
P2093
Eka Lepsveridze
Elena A Kharlamov
Kevin M Kelly
P304
P356
10.1016/J.NBD.2010.03.012
P577
2010-03-18T00:00:00Z