Prostate-specific G-protein-coupled receptor collaborates with loss of PTEN to promote prostate cancer progression. - Wikidata - wikimedia - TriplyDB

Prostate-specific G-protein-coupled receptor collaborates with loss of PTEN to promote prostate cancer progression.

Prostate-specific G-protein-coupled receptor collaborates with loss of PTEN to promote prostate cancer progression.

http://www.wikidata.org/entity/Q43124532

about

Identification of shared and unique susceptibility pathways among cancers of the lung, breast, and prostate from genome-wide association studies and tissue-specific protein interactions.Timing of the loss of Pten protein determines disease severity in a mouse model of myeloid malignancy.Rac1-mediated membrane raft localization of PI3K/p110β is required for its activation by GPCRs or PTEN loss JARID2 promotes invasion and metastasis of hepatocellular carcinoma by facilitating epithelial-mesenchymal transition through PTEN/AKT signaling.Inhibiting G protein βγ signaling blocks prostate cancer progression and enhances the efficacy of paclitaxel.Structurally related odorant ligands of the olfactory receptor OR51E2 differentially promote metastasis emergence and tumor growth Gallein, a Gβγ subunit signalling inhibitor, inhibits metastatic spread of tumour cells expressing OR51E2 and exposed to its odorant ligand.A Testosterone Metabolite 19-Hydroxyandrostenedione Induces Neuroendocrine Trans-Differentiation of Prostate Cancer Cells via an Ectopic Olfactory Receptor.

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Prostate-specific G-protein-coupled receptor collaborates with loss of PTEN to promote prostate cancer progression.

http://www.wikidata.org/entity/Q43124532

description

2015 nî lūn-bûn

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Prostate-specific G-protein-co ...... e prostate cancer progression.

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Prostate-specific G-protein-co ...... e prostate cancer progression.

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Prostate-specific G-protein-co ...... e prostate cancer progression.

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Prostate-specific G-protein-co ...... e prostate cancer progression.

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P2860

Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

Pten dose dictates cancer progression in the prostate

FISH analysis of 107 prostate cancers shows that PTEN genomic deletion is associated with poor clinical outcome.

NF-kappaB activation in human prostate cancer: important mediator or epiphenomenon?

The cBio cancer genomics portal: an open platform for exploring multidimensional cancer genomics data

Integrative analysis of complex cancer genomics and clinical profiles using the cBioPortal

NFκB signalling is upregulated in a subset of castrate-resistant prostate cancer patients and correlates with disease progression

Prostate-specific deletion of the murine Pten tumor suppressor gene leads to metastatic prostate cancer

Regulation of androgen action

The mutational landscape of lethal castration-resistant prostate cancer

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