about
Antisense oligonucleotides against rat brain alpha1E DNA and its atrial homologue decrease T-type calcium current in atrial myocytesAltered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channelsCalcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice.Antisense oligonucleotides against alpha1E reduce R-type calcium currents in cerebellar granule cells.Presynaptic homeostasis at CNS nerve terminals compensates for lack of a key Ca2+ entry pathway.Neuronal voltage-activated calcium channels: on the roles of the alpha 1E and beta 3 subunits.T-type current modulation by the actin-binding protein Kelch-like 1.The Kelch-like protein 1 modulates P/Q-type calcium current density.Altered frequency-dependent inactivation and steady-state inactivation of polyglutamine-expanded alpha1A in SCA6.Presynaptic Ca2+ channels compete for channel type-preferring slots in altered neurotransmission arising from Ca2+ channelopathy.alpha- and betaCaMKII. Inverse regulation by neuronal activity and opposing effects on synaptic strength.Ca2+ sparks and cellular distribution of ryanodine receptors in developing cardiomyocytes from rat.Insulin-mediated upregulation of T-type Ca2+ currents in GH3 cells is mediated by increased endosomal recycling and incorporation of surface membrane Cav3.1 channels.Forced-exercise delays neuropathic pain in experimental diabetes: effects on voltage-activated calcium channels.15th Annual Spring Brain Conference, March 10-13, 2004, summary report.
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P50
description
hulumtuese
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հետազոտող
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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Erika Piedras-renteria
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