RTN1-C mediates cerebral ischemia/reperfusion injury via ER stress and mitochondria-associated apoptosis pathways.
about
The Renin-Angiotensin-Aldosterone System as a Therapeutic Target in Late Injury Caused by Ischemia-Reperfusion.Melatonin Protects Against Neuronal Apoptosis via Suppression of the ATF6/CHOP Pathway in a Rat Model of Intracerebral HemorrhageHydrogen Sulfide Ameliorates Blood-Spinal Cord Barrier Disruption and Improves Functional Recovery by Inhibiting Endoplasmic Reticulum Stress-Dependent Autophagy
P2860
RTN1-C mediates cerebral ischemia/reperfusion injury via ER stress and mitochondria-associated apoptosis pathways.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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2017年學術文章
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name
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@en
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
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type
label
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@en
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@nl
prefLabel
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@en
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@nl
P2093
P2860
P356
P1476
RTN1-C mediates cerebral ische ...... associated apoptosis pathways.
@en
P2093
P2860
P356
10.1038/CDDIS.2017.465
P577
2017-10-05T00:00:00Z