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Sun Exposure and Its Effects on Human Health: Mechanisms through Which Sun Exposure Could Reduce the Risk of Developing Obesity and Cardiometabolic DysfunctionVitamin D and immunityAcute erythemal ultraviolet radiation causes systemic immunosuppression in the absence of increased 25-hydroxyvitamin D3 levels in male mice.The effects of in utero vitamin D deficiency on airway smooth muscle mass and lung function.Reversible control by vitamin D of granulocytes and bacteria in the lungs of mice: an ovalbumin-induced model of allergic airway disease.Comparing the effects of sun exposure and vitamin D supplementation on vitamin D insufficiency, and immune and cardio-metabolic function: the Sun Exposure and Vitamin D Supplementation (SEDS) Study.Can skin exposure to sunlight prevent liver inflammation?Vitamin D and allergic airway disease shape the murine lung microbiome in a sex-specific manner.Dietary Vitamin D Increases Percentages and Function of Regulatory T Cells in the Skin-Draining Lymph Nodes and Suppresses Dermal Inflammation.A pathogenic role for the integrin CD103 in experimental allergic airways disease.Response to the Boucher et al. Comments on Fleury et al. Sun Exposure and Its Effects on Human Health: Mechanisms through Which Sun Exposure Could Reduce the Risk of Developing Obesity and Cardiometabolic Dysfunction. Int. J. Environ. Res. Public HeImmune-modifying properties of topical vitamin D: Focus on dendritic cells and T cells.UV exposure and protection against allergic airways disease.Vitamin D deficiency causes airway hyperresponsiveness, increases airway smooth muscle mass, and reduces TGF-β expression in the lungs of female BALB/c mice.Modulation of the immune system by UV radiation: more than just the effects of vitamin D?Ultraviolet irradiation of mice reduces the competency of bone marrow-derived CD11c+ cells via an indomethacin-inhibitable pathway.Identification of genes differentially regulated by vitamin D deficiency that alter lung pathophysiology and inflammation in allergic airways disease.Ultraviolet radiation, vitamin D and the development of obesity, metabolic syndrome and type-2 diabetes.Toward homeostasis: regulatory dendritic cells from the bone marrow of mice with inflammation of the airways and peritoneal cavity.Clinical, Research, and Public Health Implications of Poor Measurement of Vitamin D Status.High-Dose Intramuscular Vitamin D Provides Long-Lasting Moderate Increases in Serum 25-Hydroxvitamin D Levels and Shorter-Term Changes in Plasma Calcium.Vitamin D supplementation of initially vitamin D-deficient mice diminishes lung inflammation with limited effects on pulmonary epithelial integrity.Gene regulation by 1,25-dihydroxyvitamin D3 in CD4+CD25+ cells is enabled by IL-2.1,25-dihydroxyvitamin D3 enhances the ability of transferred CD4+ CD25+ cells to modulate T helper type 2-driven asthmatic responses.Vitamin D deficiency causes deficits in lung function and alters lung structure.Mixed infection with multiple strains of murine cytomegalovirus occurs following simultaneous or sequential infection of immunocompetent mice.Prostaglandin E2 imprints a long-lasting effect on dendritic cell progenitors in the bone marrow.Primary defect in UVB-induced systemic immunomodulation does not relate to immature or functionally impaired APCs in regional lymph nodes.Topically applied 1,25-dihydroxyvitamin D3 enhances the suppressive activity of CD4+CD25+ cells in the draining lymph nodes.Investigating the roles of regulatory T cells, mast cells and interleukin-9 in the control of skin inflammation by vitamin D.Low maternal serum vitamin D during pregnancy and the risk for postpartum depression symptoms.Characterization of regulatory dendritic cells differentiated from the bone marrow of UV-irradiated mice.Altered immunity and dendritic cell activity in the periphery of mice after long-term engraftment with bone marrow from ultraviolet-irradiated mice.Sub-erythemal ultraviolet radiation reduces metabolic dysfunction in already overweight mice.Ultraviolet radiation suppresses obesity and symptoms of metabolic syndrome independently of vitamin D in mice fed a high-fat diet.Reduced immune responses in chimeric mice engrafted with bone marrow cells from mice with airways inflammation.IFNβ inhibits the development of allergen tolerance and is conducive to the development of asthma on subsequent allergen exposure.Transmission of two Australian strains of murine cytomegalovirus (MCMV) in enclosure populations of house mice (Mus domesticus).Suppression of the asthmatic phenotype by ultraviolet B-induced, antigen-specific regulatory cellsMaternal high fat diet compromises survival and modulates lung development of offspring, and impairs lung function of dams (female mice)
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description
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wetenschapper
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հետազոտող
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name
Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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Shelley Gorman
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P108
P1053
R-9953-2017
P106
P1153
8621466800
P31
P3829
P496
0000-0002-7111-6735