Early effects of doxorubicin in perfused heart: transcriptional profiling reveals inhibition of cellular stress response genes.
about
Cardiac systolic dysfunction in doxorubicin-challenged rats is associated with upregulation of MuRF2 and MuRF3 E3 ligasesAutophagy and mitophagy in the context of doxorubicin-induced cardiotoxicity.Exposing cardiomyocytes to subclinical concentrations of doxorubicin rapidly reduces their creatine transport.Doxorubicin increases oxidative metabolism in HL-1 cardiomyocytes as shown by 13C metabolic flux analysis.Aspartate facilitates mitochondrial function, growth arrest and survival during doxorubicin exposure.Signalling mechanisms underlying doxorubicin and Nox2 NADPH oxidase-induced cardiomyopathy: involvement of mitofusin-2.FGF-2 and FGF-16 protect isolated perfused mouse hearts from acute doxorubicin-induced contractile dysfunction.
P2860
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P2860
Early effects of doxorubicin in perfused heart: transcriptional profiling reveals inhibition of cellular stress response genes.
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2010 nî lūn-bûn
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Early effects of doxorubicin i ...... ellular stress response genes.
@en
Early effects of doxorubicin i ...... ellular stress response genes.
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type
label
Early effects of doxorubicin i ...... ellular stress response genes.
@en
Early effects of doxorubicin i ...... ellular stress response genes.
@nl
prefLabel
Early effects of doxorubicin i ...... ellular stress response genes.
@en
Early effects of doxorubicin i ...... ellular stress response genes.
@nl
P2093
P2860
P50
P1476
Early effects of doxorubicin i ...... cellular stress response genes
@en
P2093
Laurence Kay
Malgorzata Tokarska-Schlattner
Rita Guzun
Séverine Gratia
Valdur Saks
P2860
P304
P356
10.1152/AJPREGU.00360.2009
P577
2010-01-06T00:00:00Z