Ras-induced reactive oxygen species promote growth factor-independent proliferation in human CD34+ hematopoietic progenitor cells.
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To breathe or not to breathe: the haematopoietic stem/progenitor cells dilemmaRedox regulation of stem/progenitor cells and bone marrow nicheLive imaging of innate immune cell sensing of transformed cells in zebrafish larvae: parallels between tumor initiation and wound inflammationEltrombopag modulates reactive oxygen species and decreases acute myeloid leukemia cell survivalNovel roles of reactive oxygen species in the pathogenesis of acute myeloid leukemia.Gene therapy of chronic granulomatous disease: the engraftment dilemmaCellular redox imbalance and changes of protein S-glutathionylation patterns are associated with senescence induced by oncogenic H-rasRole of reactive oxygen species in the radiation response of human hematopoietic stem/progenitor cells.Myeloid cell-derived reactive oxygen species externally regulate the proliferation of myeloid progenitors in emergency granulopoiesisHydrogen peroxide fuels aging, inflammation, cancer metabolism and metastasis: the seed and soil also needs "fertilizer".GnRH Regulates Gonadotropin Gene Expression Through NADPH/Dual Oxidase-Derived Reactive Oxygen Species.The mitochondrial protein C1qbp promotes cell proliferation, migration and resistance to cell death.The glutathione disulfide mimetic NOV-002 inhibits cyclophosphamide-induced hematopoietic and immune suppression by reducing oxidative stress.Normal Hematopoietic Progenitor Subsets Have Distinct Reactive Oxygen Species, BCL2 and Cell-Cycle Profiles That Are Decoupled from Maturation in Acute Myeloid LeukemiaProstaglandin E2 promotes proliferation of skeletal muscle myoblasts via EP4 receptor activation.Activated Oncogenic Pathway Modifies Iron Network in Breast Epithelial Cells: A Dynamic Modeling Perspective.Novel oxidatively activated agents modify DNA and are enhanced by ercc1 silencing.Redox control of leukemia: from molecular mechanisms to therapeutic opportunities.BCL-2 inhibition targets oxidative phosphorylation and selectively eradicates quiescent human leukemia stem cellsγ-Catenin is overexpressed in acute myeloid leukemia and promotes the stabilization and nuclear localization of β-cateninReactive oxygen species in eradicating acute myeloid leukemic stem cellsHydrogen peroxide (H2O2) induces leukemic but not normal hematopoietic cell death in a dose-dependent manner.Neurofibromin-deficient myeloid cells are critical mediators of aneurysm formation in vivo.Stem cells, redox signaling, and stem cell aging.The sweet taste of death: glucose triggers apoptosis during yeast chronological aging.Optimizing therapy for iron overload in the myelodysplastic syndromes: recent developments.Oxygen regulates molecular mechanisms of cancer progression and metastasis.DNA damage accumulation and repair defects in acute myeloid leukemia: implications for pathogenesis, disease progression, and chemotherapy resistance.Therapeutically targeting SELF-reinforcing leukemic niches in acute myeloid leukemia: A worthy endeavor?Hematopoietic stem/progenitor cells express myoglobin and neuroglobin: adaptation to hypoxia or prevention from oxidative stress?KRAS(G12V) enhances proliferation and initiates myelomonocytic differentiation in human stem/progenitor cells via intrinsic and extrinsic pathways.Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans.Reactive Oxygen Species-Producing Myeloid Cells Act as a Bone Marrow Niche for Sterile Inflammation-Induced Reactive Granulopoiesis.Effects of mitochondrial ferritin overexpression in normal and sideroblastic erythroid progenitors.PKC epsilon is a Key Regulator of Mitochondrial Redox Homeostasis in Acute Myeloid Leukemia.Reactive oxygen species (ROS) are a key determinant of cancer's metabolic phenotype.RUNX-mediated growth arrest and senescence are attenuated by diverse mechanisms in cells expressing RUNX1 fusion oncoproteins.Oxidative stress response induced by chemotherapy in leukemia treatment.Reactive oxygen species in haematopoiesis: leukaemic cells take a walk on the wild side.
P2860
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P2860
Ras-induced reactive oxygen species promote growth factor-independent proliferation in human CD34+ hematopoietic progenitor cells.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@en
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@nl
type
label
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@en
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@nl
prefLabel
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@en
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@nl
P2093
P1433
P1476
Ras-induced reactive oxygen sp ...... ematopoietic progenitor cells.
@en
P2093
Alan K Burnett
Alex Tonks
Lorna Pearn
Paul S Hole
Philip E James
Richard L Darley
P304
P356
10.1182/BLOOD-2009-06-222869
P407
P577
2009-12-10T00:00:00Z