Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
about
Glutathione peroxidase 7 protects against oxidative DNA damage in oesophageal cellsLocation-specific epigenetic regulation of the metallothionein 3 gene in esophageal adenocarcinomas.DNA hypermethylation regulates the expression of members of the Mu-class glutathione S-transferases and glutathione peroxidases in Barrett's adenocarcinoma.Hypermethylation and loss of expression of glutathione peroxidase-3 in Barrett's tumorigenesis.Barrett's oesophagus.Role of intracellular calcium and NADPH oxidase NOX5-S in acid-induced DNA damage in Barrett's cells and Barrett's esophageal adenocarcinoma cells.Evidence for DNA damage checkpoint activation in barrett esophagusInflammatory mediators in gastroesophageal reflux disease: impact on esophageal motility, fibrosis, and carcinogenesis.Long-term results of ablation with antireflux surgery for Barrett's esophagus: a clinical and molecular biologic study.The inhibitory effect of PIK-75 on inflammatory mediator response induced by hydrogen peroxide in feline esophageal epithelial cellsInflammation and oxidative stress markers and esophageal adenocarcinoma incidence in a Barrett's esophagus cohort.Free radicals and antioxidant systems in reflux esophagitis and Barrett's esophagus.Signaling in H2O2-induced increase in cell proliferation in Barrett's esophageal adenocarcinoma cells.Role of Rac1 in regulation of NOX5-S function in Barrett's esophageal adenocarcinoma cells.The Protective Effect of Quercetin-3-O-β-D-Glucuronopyranoside on Ethanol-induced Damage in Cultured Feline Esophageal Epithelial Cells.Rho Kinase ROCK2 Mediates Acid-Induced NADPH Oxidase NOX5-S Expression in Human Esophageal Adenocarcinoma Cells.Bile acids in combination with low pH induce oxidative stress and oxidative DNA damage: relevance to the pathogenesis of Barrett's oesophagus.The Protective Effect of Eupatilin against Hydrogen Peroxide-Induced Injury Involving 5-Lipoxygenase in Feline Esophageal Epithelial Cells.Increasing expression of hypoxia-inducible proteins in the Barrett's metaplasia-dysplasia-adenocarcinoma sequence.Differential expression of Hsp27 in normal oesophagus, Barrett's metaplasia and oesophageal adenocarcinomas.Role of NADPH oxidase NOX5-S, NF-κB, and DNMT1 in acid-induced p16 hypermethylation in Barrett's cellsInhibitory effect of FSLLRY-NH2 on inflammatory responses induced by hydrogen peroxide in HepG2 cells.The protective mechanism of QGC in feline esophageal epithelial cells by interleukin-1β treatment.Synthesis and characterization of anti-EGFR fluorescent nanoparticles for optical molecular imaging.The protective mechanism of quercetin-3-O-β-D-glucuronopyranoside (QGC) in H2O2-induced injury of feline esophageal epithelial cells.Bile acid reflux contributes to development of esophageal adenocarcinoma via activation of phosphatidylinositol-specific phospholipase Cgamma2 and NADPH oxidase NOX5-S.Role of a novel bile acid receptor TGR5 in the development of oesophageal adenocarcinoma.Mechanisms of oxidant production in esophageal squamous cell and Barrett's cell lines.STAT5 mediates PAF-induced NADPH oxidase NOX5-S expression in Barrett's esophageal adenocarcinoma cells.Esophageal acid exposure at pH < or = 2 is more common in Barrett's esophagus patients and is associated with oxidative stress.Increased Oxidative Stress in the Proximal Stomach of Patients with Barrett's Esophagus and Adenocarcinoma of the Esophagus and Esophagogastric Junction.Superoxide anions produced by inflammatory cells play an important part in the pathogenesis of acid and pepsin induced oesophagitis in rabbits.Intrinsic resistance triggered under acid loading within normal esophageal epithelial cells: NHE1- and ROS-mediated survival.Superoxide dismutase prevents development of adenocarcinoma in a rat model of Barrett's esophagus.Reactive oxygen species and chemokines: are they elevated in the esophageal mucosa of children with gastroesophageal reflux disease?Inflammation and oxidative stress in gastroesophageal reflux disease.The role of oxidative DNA damage, DNA repair, GSTM1, SOD2 and OGG1 polymorphisms in individual susceptibility to Barrett's esophagus.Glutathione peroxidase isoforms as part of the local antioxidative defense system in normal and Barrett's esophagus.H(2)O(2): a mediator of esophagitis-induced damage to calcium-release mechanisms in cat lower esophageal sphincter.Vegetarianism as a protective factor for reflux esophagitis: a retrospective, cross-sectional study between Buddhist priests and general population.
P2860
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P2860
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh-hant
name
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@en
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@nl
type
label
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@en
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@nl
prefLabel
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@en
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@nl
P2093
P2860
P356
P1433
P1476
Mucosal reactive oxygen species production in oesophagitis and Barrett's oesophagus.
@en
P2093
Eiznhamer D
Keshavarzian A
Mobarhan S
P2860
P304
P356
10.1136/GUT.37.2.168
P407
P577
1995-08-01T00:00:00Z