Theaflavins target Fas/caspase-8 and Akt/pBad pathways to induce apoptosis in p53-mutated human breast cancer cells. - Wikidata - wikimedia - TriplyDB

Theaflavins target Fas/caspase-8 and Akt/pBad pathways to induce apoptosis in p53-mutated human breast cancer cells.

Theaflavins target Fas/caspase-8 and Akt/pBad pathways to induce apoptosis in p53-mutated human breast cancer cells.

http://www.wikidata.org/entity/Q43227672

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MicroRNA-122 regulates caspase-8 and promotes the apoptosis of mouse cardiomyocytes.Green tea polyphenols induce p53-dependent and p53-independent apoptosis in prostate cancer cells through two distinct mechanisms Curcumin enhances the efficacy of chemotherapy by tailoring p65NFκB-p300 cross-talk in favor of p53-p300 in breast cancer.Black tea polyphenols inhibit tumor proteasome activity Aspirin inhibits epithelial-to-mesenchymal transition and migration of oncogenic K-ras-expressing non-small cell lung carcinoma cells by down-regulating E-cadherin repressor Slug.Inhibitory Effects of the Four Main Theaflavin Derivatives Found in Black Tea on Ovarian Cancer Cells Impaired mitochondrial metabolism and mammary carcinogenesis Theaflavin-3, 3'-digallate induces apoptosis and G2 cell cycle arrest through the Akt/MDM2/p53 pathway in cisplatin-resistant ovarian cancer A2780/CP70 cells.Targeting the Fas/FasL signaling pathway in cancer therapy.Restoration of tumor suppressor p53 by differentially regulating pro- and anti-p53 networks in HPV-18-infected cervical cancer cells.Perforin-dependent direct cytotoxicity in natural killer cells induces considerable knockdown of spontaneous lung metastases and computer modelling-proven tumor cell dormancy in a HT29 human colon cancer xenograft mouse model.Targeting RET to induce medullary thyroid cancer cell apoptosis: an antagonistic interplay between PI3K/Akt and p38MAPK/caspase-8 pathways.Crocetin exploits p53-induced death domain (PIDD) and FAS-associated death domain (FADD) proteins to induce apoptosis in colorectal cancer.Effect of methylprednisolone on the activities of caspase-3, -6, -8 and -9 in rabbits with acute spinal cord injury.Inhibitory effect of black tea pigments, theaflavin‑3/3'-gallate against cisplatin-resistant ovarian cancer cells by inducing apoptosis and G1 cell cycle arrest.Theaflavins suppress tumor growth and metastasis via the blockage of the STAT3 pathway in hepatocellular carcinoma.Pre-treated theaflavin-3,3'-digallate has a higher inhibitory effect on the HCT116 cell line.Polyphenol supplementation alters intramuscular apoptotic signaling following acute resistance exercise.Hepatocellular apoptosis in mice is associated with early upregulation of mitochondrial glucose metabolism.

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Theaflavins target Fas/caspase-8 and Akt/pBad pathways to induce apoptosis in p53-mutated human breast cancer cells.

http://www.wikidata.org/entity/Q43227672

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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type

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Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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Theaflavins target Fas/caspase ...... ted human breast cancer cells.

@en

Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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Theaflavins target Fas/caspase ...... ted human breast cancer cells.

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Arghya Adhikary

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Baisakhi Saha

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Dewan Md Sakib Hossain

http://www.w3.org/2001/XMLSchema#string

Gaurisankar Sa

http://www.w3.org/2001/XMLSchema#string

Juni Chakraborty

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Kaushik Das

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Lakshmishri Lahiry

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Shuvomoy Banerjee

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Suchismita Mohanty

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Tanya Das

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P2860

Bcl-x(L) sequesters its C-terminal membrane anchor in soluble, cytosolic homodimers

The phosphoinositide 3-kinase pathway

Mammalian caspases: structure, activation, substrates, and functions during apoptosis

Retinoblastoma protein modulates gankyrin-MDM2 in regulation of p53 stability and chemosensitivity in cancer cells

Death receptors: signaling and modulation

Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

Bid participates in genotoxic drug-induced apoptosis of HeLa cells and is essential for death receptor ligands' apoptotic and synergistic effects

Fas ligand-independent, FADD-mediated activation of the Fas death pathway by anticancer drugs.

Bcl-XL inhibits membrane permeabilization by competing with Bax.

Fas ligand-induced apoptosis.

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259-268

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scholarly article

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10.1093/CARCIN/BGP240

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2

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31

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2009-12-07T00:00:00Z

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19969555

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