NK cells: a double-edged sword in chronic hepatitis B virus infection. - Wikidata - wikimedia - TriplyDB

NK cells: a double-edged sword in chronic hepatitis B virus infection.

NK cells: a double-edged sword in chronic hepatitis B virus infection.

http://www.wikidata.org/entity/Q43237496

about

Recognition and Regulation of T Cells by NK Cells Hepatitis B virus and microRNAs: Complex interactions affecting hepatitis B virus replication and hepatitis B virus-associated diseases Current and future antiviral drug therapies of hepatitis B chronic infection A Practical Model Evaluating Antiviral Cytokines by Natural Killer Cells in Treatment Naïve Patients with Chronic Hepatitis B Virus Infection.Downregulation of interleukin-18-mediated cell signaling and interferon gamma expression by the hepatitis B virus e antigen.NK cells and virus-related cancers.Postexposure prophylactic effect of hepatitis B virus (HBV)-active antiretroviral therapy against HBV infection.Defective natural killer cell anti-viral capacity in paediatric HBV infection.Gut microbiota modulate the immune effect against hepatitis B virus infection.Altered Immune Profiles of Natural Killer Cells in Chronic Hepatitis B Patients: A Systematic Review and Meta-Analysis.Potential role of killer immunoglobulin receptor genes among individuals vaccinated against hepatitis B virus in Lebanon.T Cells Infiltrating Diseased Liver Express Ligands for the NKG2D Stress Surveillance System.Viral (hepatitis C virus, hepatitis B virus, HIV) persistence and immune homeostasis.The immune tolerant phase of chronic HBV infection: new perspectives on an old concept Age-Dependent Immune Events during HBV Infection from Birth to Adulthood: An Alternative Interpretation.HBV and the immune response.Natural history of chronic hepatitis B virus infection.Host-virus interactions in hepatitis B and hepatitis C infection.Natural killer cell phenotype modulation and natural killer/T-cell interplay in nucleos(t)ide analogue-treated hepatitis e antigen-negative patients with chronic hepatitis B.Extracellular Vesicles Deliver Host and Virus RNA and Regulate Innate Immune Response Hepatitis B virus-triggered autophagy targets TNFRSF10B/death receptor 5 for degradation to limit TNFSF10/TRAIL response.Immune Evasion Strategies during Chronic Hepatitis B and C Virus Infection Extracellular Vesicles Including Exosomes Regulate Innate Immune Responses to Hepatitis B Virus Infection.Tolerance and immunity to pathogens in early life: insights from HBV infection.CXCR6 marks a novel subset of T-bet(lo)Eomes(hi) natural killer cells residing in human liver.Clinical Significance of Quantitative HBsAg Titres and its Correlation With HBV DNA Levels in the Natural History of Hepatitis B Virus Infection.Indoleamine-2,3-dioxygenase as an effector and an indicator of protective immune responses in patients with acute hepatitis B.A potent human neutralizing antibody Fc-dependently reduces established HBV infections Circulating CD56dim natural killer cells and CD56+ T cells that produce interferon-γ or interleukin-10 are expanded in asymptomatic, E antigen-negative patients with persistent hepatitis B virus infection.Immunotherapy for Chronic Hepatitis B Virus Infection.Hepatitis B virus-specific T cells associate with viral control upon nucleos(t)ide-analogue therapy discontinuation.Global strategies are required to cure and eliminate HBV infection.Tissue-resident Eomes(hi) T-bet(lo) CD56(bright) NK cells with reduced proinflammatory potential are enriched in the adult human liver.Changes in natural killer cells and exhausted memory regulatory T Cells with corticosteroid therapy in acute autoimmune hepatitis.The Murine Natural Cytotoxic Receptor NKp46/NCR1 Controls TRAIL Protein Expression in NK Cells and ILC1s.Is it possible to stop nucleos(t)ide analogue treatment in chronic hepatitis B patients?Decreased Siglec-9 Expression on Natural Killer Cell Subset Associated With Persistent HBV Replication.Immunogenicity of adenovirus-vector vaccine targeting hepatitis B virus: non-clinical safety assessment in non-human primates

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NK cells: a double-edged sword in chronic hepatitis B virus infection.

http://www.wikidata.org/entity/Q43237496

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-my

2013年学术文章

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2013年學術文章

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2013年學術文章

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name

NK cells: a double-edged sword in chronic hepatitis B virus infection.

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NK cells: a double-edged sword in chronic hepatitis B virus infection.

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type

label

NK cells: a double-edged sword in chronic hepatitis B virus infection.

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NK cells: a double-edged sword in chronic hepatitis B virus infection.

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NK cells: a double-edged sword in chronic hepatitis B virus infection.

@en

NK cells: a double-edged sword in chronic hepatitis B virus infection.

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FIMMU.2013.00057

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Frontiers in Immunology

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NK cells: a double-edged sword in chronic hepatitis B virus infection

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Dimitra Peppa

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P2860

Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS

Intrahepatic IL-10 maintains NKG2A+Ly49- liver NK cells in a functionally hyporesponsive state.

Blockade of immunosuppressive cytokines restores NK cell antiviral function in chronic hepatitis B virus infection

Concerted action of the FasL/Fas and perforin/granzyme A and B pathways is mandatory for the development of early viral hepatitis but not for recovery from viral infection.

Absence of mouse 2B4 promotes NK cell-mediated killing of activated CD8+ T cells, leading to prolonged viral persistence and altered pathogenesis.

Hydrodynamic injection of viral DNA: a mouse model of acute hepatitis B virus infection

HIV-1 adaptation to NK-cell-mediated immune pressure

TGF-β1 down-regulation of NKG2D/DAP10 and 2B4/SAP expression on human NK cells contributes to HBV persistence

Upregulation of the Tim-3/galectin-9 pathway of T cell exhaustion in chronic hepatitis B virus infection.

CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection.

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57

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