Novel potassium channel blocker, 4-AP-3-MeOH, inhibits fast potassium channels and restores axonal conduction in injured guinea pig spinal cord white matter.
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Real-time CARS imaging reveals a calpain-dependent pathway for paranodal myelin retraction during high-frequency stimulationMolecular mechanisms of acrolein-mediated myelin destruction in CNS trauma and diseaseAcrolein-mediated conduction loss is partially restored by K⁺ channel blockersAcrolein as a novel therapeutic target for motor and sensory deficits in spinal cord injuryAcrolein detection: potential theranostic utility in multiple sclerosis and spinal cord injury.Paranodal myelin damage after acute stretch in Guinea pig spinal cordAn overview of pharmacological approaches for management and repair of spinal cord injuries.Acrolein-mediated injury in nervous system trauma and diseasesCervical spinal demyelination with ethidium bromide impairs respiratory (phrenic) activity and forelimb motor behavior in rats.New insights in the pathogenesis of multiple sclerosis--role of acrolein in neuronal and myelin damage.Potassium channel blockers as an effective treatment to restore impulse conduction in injured axons.Ion channel blockers and spinal cord injury.From demyelination to remyelination: the road toward therapies for spinal cord injury.CNS repair and axon regeneration: Using genetic variation to determine mechanisms.The inhibition by di(2-ethylhexyl)-phthalate of erg-mediated K⁺ current in pituitary tumor (GH₃) cells.Restoration of Visual Function by Enhancing Conduction in Regenerated AxonsSubtle paranodal injury slows impulse conduction in a mathematical model of myelinated axons.Potassium channel blocker, 4-aminopyridine-3-methanol, restores axonal conduction in spinal cord of an animal model of multiple sclerosis.Computer modeling of mild axonal injury: implications for axonal signal transmission.Primary paranode demyelination modulates slowly developing axonal depolarization in a model of axonal injury.The effects of paranodal myelin damage on action potential depend on axonal structure.A Sensitized IGF1 Treatment Restores Corticospinal Axon-Dependent Functions.Potassium Channels: A Potential Therapeutic Target for Parkinson's Disease.Reduced conduction failure of the main axon of polymodal nociceptive C-fibres contributes to painful diabetic neuropathy in rats.Parallel evaluation of two potassium channel blockers in restoring conduction in mechanical spinal cord injury in rat.Potassium channel blockers restore axonal conduction in CNS trauma and diseases.
P2860
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P2860
Novel potassium channel blocker, 4-AP-3-MeOH, inhibits fast potassium channels and restores axonal conduction in injured guinea pig spinal cord white matter.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
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2009年学术文章
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2009年學術文章
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2009年學術文章
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name
Novel potassium channel blocke ...... pig spinal cord white matter.
@en
Novel potassium channel blocke ...... pig spinal cord white matter.
@nl
type
label
Novel potassium channel blocke ...... pig spinal cord white matter.
@en
Novel potassium channel blocke ...... pig spinal cord white matter.
@nl
prefLabel
Novel potassium channel blocke ...... pig spinal cord white matter.
@en
Novel potassium channel blocke ...... pig spinal cord white matter.
@nl
P2093
P2860
P356
P1476
Novel potassium channel blocke ...... pig spinal cord white matter.
@en
P2093
Daniel Smith
Ji-Xin Cheng
Richard Borgens
Steven Bryn
Wenjing Sun
P2860
P304
P356
10.1152/JN.00154.2009
P407
P577
2009-11-18T00:00:00Z