Extracellular L-arginine is required for optimal NO synthesis by eNOS and iNOS in the rat mesenteric artery wall.
about
Loss of p53 in stromal fibroblasts promotes epithelial cell invasion through redox-mediated ICAM1 signal.Extracellular arginine rapidly dilates in vivo intestinal arteries and arterioles through a nitric oxide mechanism.ATP inhibits pump activity of lymph vessels via adenosine A1 receptor-mediated involvement of NO- and ATP-sensitive K+ channels.Transport of extracellular l-arginine via cationic amino acid transporter is required during in vivo endothelial nitric oxide production.Stromelysin-1 5A/6A and eNOS T-786C polymorphisms, MTHFR C677T and A1298C mutations, and cigarette-cannabis smoking: a pilot, hypothesis-generating study of gene-environment pathophysiological associations with Buerger's disease.Functional status of microvascular vasomotion is impaired in spontaneously hypertensive rat.
P2860
Extracellular L-arginine is required for optimal NO synthesis by eNOS and iNOS in the rat mesenteric artery wall.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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name
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@en
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@nl
type
label
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@en
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@nl
prefLabel
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@en
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@nl
P2860
P356
P1476
Extracellular L-arginine is re ...... he rat mesenteric artery wall.
@en
P2093
Andrew MacKenzie
Roger M Wadsworth
P2860
P304
P356
10.1038/SJ.BJP.0705380
P407
P577
2003-08-01T00:00:00Z