about
Caspr3 and caspr4, two novel members of the caspr family are expressed in the nervous system and interact with PDZ domainsThe Myelin and Lymphocyte Protein MAL Is Required for Binding and Activity of Clostridium perfringens ε-ToxinMyelin-associated glycoprotein gene mutation causes Pelizaeus-Merzbacher disease-like disorderClaudin-1 induced sealing of blood-brain barrier tight junctions ameliorates chronic experimental autoimmune encephalomyelitisReversal of non-hydroxy:alpha-hydroxy galactosylceramide ratio and unstable myelin in transgenic mice overexpressing UDP-galactose:ceramide galactosyltransferase.MAL overexpression leads to disturbed expression of genes that influence cytoskeletal organization and differentiation of Schwann cells.An essential role of MAG in mediating axon-myelin attachment in Charcot-Marie-Tooth 1A diseaseNormal-appearing white matter in multiple sclerosis is in a subtle balance between inflammation and neuroprotection.Clostridium perfringens Epsilon Toxin Causes Selective Death of Mature Oligodendrocytes and Central Nervous System Demyelination.Interleukin-22 is increased in multiple sclerosis patients and targets astrocytesVIP17/MAL expression modulates epithelial cyst formation and ciliogenesisThe raft-associated protein MAL is required for maintenance of proper axon--glia interactions in the central nervous system.Transcriptional regulation induced by cAMP elevation in mouse Schwann cellsLoss of Myelin Basic Protein Function Triggers Myelin Breakdown in Models of Demyelinating Diseases.Cocapture of cognate and bystander antigens can activate autoreactive B cells.DARC shuttles inflammatory chemokines across the blood-brain barrier during autoimmune central nervous system inflammation.Molecular changes in normal appearing white matter in multiple sclerosis are characteristic of neuroprotective mechanisms against hypoxic insult.Gene expression profiling in nerve biopsy of vasculitic neuropathy.The immunomodulatory oligodendrocyte.Identification of Inflammatory, Metabolic, and Cell Survival Pathways Contributing to Cerebral Small Vessel Disease by Postmortem Gene Expression Microarray.Septin/anillin filaments scaffold central nervous system myelin to accelerate nerve conduction.Selective in vivo removal of pathogenic anti-MAG autoantibodies, an antigen-specific treatment option for anti-MAG neuropathy.
P50
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P50
description
onderzoeker
@nl
name
Nicole Schaeren-Wiemers
@ast
Nicole Schaeren-Wiemers
@en
Nicole Schaeren-Wiemers
@es
Nicole Schaeren-Wiemers
@nl
Nicole Schaeren-Wiemers
@sl
type
label
Nicole Schaeren-Wiemers
@ast
Nicole Schaeren-Wiemers
@en
Nicole Schaeren-Wiemers
@es
Nicole Schaeren-Wiemers
@nl
Nicole Schaeren-Wiemers
@sl
prefLabel
Nicole Schaeren-Wiemers
@ast
Nicole Schaeren-Wiemers
@en
Nicole Schaeren-Wiemers
@es
Nicole Schaeren-Wiemers
@nl
Nicole Schaeren-Wiemers
@sl