Effect of chronic analgesic exposure on the central serotonin system: a possible mechanism of analgesic abuse headache. - Wikidata - wikimedia - TriplyDB

Effect of chronic analgesic exposure on the central serotonin system: a possible mechanism of analgesic abuse headache.

Effect of chronic analgesic exposure on the central serotonin system: a possible mechanism of analgesic abuse headache.

http://www.wikidata.org/entity/Q43503975

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2002 Wolff Award. 5 -HT2A receptor activation and nitric oxide synthesis: a possible mechanism determining migraine attacks.Abnormal cortical responses to somatosensory stimulation in medication-overuse headache.Do butalbital-containing products have a role in the management of migraine?Habituation to pain in "medication overuse headache": a CO2 laser-evoked potential study.Medication-overuse headache and opioid-induced hyperalgesia: A review of mechanisms, a neuroimmune hypothesis and a novel approach to treatment.Medication overuse headache: history, features, prevention and management strategies.Chronic daily headache: a scientist's perspective.Chronic daily headache: a clinician's perspective.Mechanisms of non-opioid analgesics beyond cyclooxygenase enzyme inhibition.Chronic daily headaches in children.General principles of migraine management: the changing role of prevention.Nerve growth factor and chronic daily headache: a potential implication for therapy.Increased activity of serotonin uptake in platelets in medication overuse headache following regular intake of analgesics and triptans Medication overuse headache: biobehavioral issues and solutions.Prevalence of medication overuse headache in an interdisciplinary pain clinic Management of medication-overuse headache.Pathophysiology of medication-overuse headache: implications from animal studies.Pathophysiology of medication overuse headache--an update.Placebo response in a patient with chronic migraine and ergotic overuse.Serotonin depletion, cortical spreading depression, and trigeminal nociception.Cortical hyperexcitability and mechanism of medication-overuse headache.Chronic daily headache prophylaxis with tizanidine: a double-blind, placebo-controlled, multicenter outcome study.Analysis of leukocytes in medication-overuse headache, chronic migraine, and episodic migraine.Modulation of central sensitisation by detoxification in MOH: results of a 12-month detoxification study.Serotonergic activity contributes to analgesic overuse in chronic tension-type headache.Rizatriptan overuse promotes hyperalgesia induced by dural inflammatory stimulation in rats by modulation of the serotonin system.Acetaminophen enhances the reflective learning process

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Effect of chronic analgesic exposure on the central serotonin system: a possible mechanism of analgesic abuse headache.

http://www.wikidata.org/entity/Q43503975

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2000 nî lūn-bûn

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P2860

Ethical guidelines for investigations of experimental pain in conscious animals

The role of 5-hydroxytryptamine (5-HT) receptor subtypes and plasticity in the 5-HT systems in the regulation of nociceptive sensitivity.

Transformed migraine, analgesic rebound, and other chronic daily headaches.

Microinjection of morphine into the periaqueductal gray evokes the release of serotonin from spinal cord.

Antinociceptive effect of ketanserin in mice: involvement of supraspinal 5-HT2 receptors in nociceptive transmission.

Lack of binding of acetaminophen to 5-HT receptor or uptake sites (or eleven other binding/uptake assays).

Localization of the central antinociceptive effects of diclofenac in the rat.

Interactions between serotonin and substance P in the spinal regulation of nociception.

Platelet serotonin in patients with analgesic-induced headache.

The antinociceptive action of paracetamol is associated with changes in the serotonergic system in the rat brain.

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