Insulin-like growth factor-I and over-expression of Bcl-xL prevent glucose-mediated apoptosis in Schwann cells.
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Diabetic gastrointestinal motility disorders and the role of enteric nervous system: current status and future directionsProtection against glucose-induced neuronal death by NAAG and GCP II inhibition is regulated by mGluR3Elevated protein carbonylation, and misfolding in sciatic nerve from db/db and Sod1(-/-) mice: plausible link between oxidative stress and demyelinationTransforming growth factor-beta induces cellular injury in experimental diabetic neuropathySchwann cell interactions with axons and microvessels in diabetic neuropathy.Tangluoning, a traditional Chinese medicine, attenuates in vivo and in vitro diabetic peripheral neuropathy through modulation of PERK/Nrf2 pathwaySensory neurons and schwann cells respond to oxidative stress by increasing antioxidant defense mechanisms.Influence of insulin-like growth factor-I (IGF-I) on nerve autografts and tissue-engineered nerve grafts.Neutralization of schwann cell-secreted VEGF is protective to in vitro and in vivo experimental diabetic neuropathy.GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathwayPuerarin prevents high glucose-induced apoptosis of Schwann cells by inhibiting oxidative stress.Behavior of solvent-exposed hydrophobic groove in the anti-apoptotic Bcl-XL protein: clues for its ability to bind diverse BH3 ligands from MD simulations.Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy.Ginsenoside Rb1 relieves glucose fluctuation-induced oxidative stress and apoptosis in Schwann cells.Degradation and dephosphorylation of focal adhesion kinase during okadaic acid-induced apoptosis in human neuroblastoma cells.Astrocyte-Specific Overexpression of Insulin-Like Growth Factor-1 Protects Hippocampal Neurons and Reduces Behavioral Deficits following Traumatic Brain Injury in Mice.The identification of gene expression profiles associated with progression of human diabetic neuropathyEffects of High Glucose on Cell Viability and Differentiation in Primary Cultured Schwann Cells: Potential Role of ERK Signaling Pathway.The effect of recombinant human growth hormone therapy in patients with completed stroke: a pilot trial.Oxidative injury and neuropathy in diabetes and impaired glucose tolerance.Protective Effect of Puerarin Against Oxidative Stress Injury of Neural Cells and Related Mechanisms.Evaluation tools and animal models of peripheral neuropathies.Erythropoietin Modification Enhances the Protection of Mesenchymal Stem Cells on Diabetic Rat-Derived Schwann Cells: Implications for Diabetic Neuropathy.Immortalized adult rodent Schwann cells as in vitro models to study diabetic neuropathy.Schwann Cell-Mediated Preservation of Vision in Retinal Degenerative Diseases via the Reduction of Oxidative Stress: A Possible Mechanism.Therapeutic augmentation of the growth hormone axis to improve outcomes following peripheral nerve injury.Protection of neuronal cell death against diabetes-induced apoptosis by Fas blocker ZB4.Loss of enteric neurons accompanied by decreased expression of GDNF and PI3K/Akt pathway in diabetic rats.Intrathecal administration of IGF-I by AAVrh10 improves sensory and motor deficits in a mouse model of diabetic neuropathy.Chinese medicine in diabetic peripheral neuropathy: experimental research on nerve repair and regeneration.Diabetic Schwann cells suffer from nerve growth factor and neurotrophin-3 underproduction and poor associability with axons.Neuritin is expressed in Schwann cells and down-regulated in apoptotic Schwann cells under hyperglycemia.Death of hypothalamic astrocytes in poorly controlled diabetic rats is associated with nuclear translocation of apoptosis inducing factor.Effect of Jinmaitong serum on the proliferation of rat Schwann cells cultured in high glucose medium.Puerarin may protect against Schwann cell damage induced by glucose fluctuation.Methylglyoxal induces apoptosis through oxidative stress-mediated activation of p38 mitogen-activated protein kinase in rat Schwann cells.Erythropoietin attenuates oxidative stress and apoptosis in Schwann cells isolated from streptozotocin-induced diabetic rats.Glutamate Inhibits the Pro-Survival Effects of Insulin-Like Growth Factor-1 on Retinal Ganglion Cells in Hypoxic Neonatal Rat Retina.Ice plant (Mesembryanthemum crystallinum) improves hyperglycaemia and memory impairments in a Wistar rat model of streptozotocin-induced diabetes.Anti-Apoptotic Effect of IGF1 on Schwann Exposed to Hyperglycemia is Mediated by Neuritin, a Novel Neurotrophic Factor.
P2860
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P2860
Insulin-like growth factor-I and over-expression of Bcl-xL prevent glucose-mediated apoptosis in Schwann cells.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@en
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@nl
type
label
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@en
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@nl
prefLabel
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@en
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@nl
P2093
P2860
P356
P1476
Insulin-like growth factor-I a ...... ed apoptosis in Schwann cells.
@en
P2093
P2860
P304
P356
10.1093/JNEN/60.2.147
P577
2001-02-01T00:00:00Z