Zinc supplementation does not attenuate alcohol-induced cerebellar Purkinje cell loss during the brain growth spurt period.
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The plausibility of maternal nutritional status being a contributing factor to the risk for fetal alcohol spectrum disorders: the potential influence of zinc status as an example.Nutrition implications for fetal alcohol spectrum disorder.Developmental alcohol exposure alters light-induced phase shifts of the circadian activity rhythm in ratsFetal alcohol spectrum disorders: experimental treatments and strategies for intervention.Antioxidants prevent ethanol-associated apoptosis in fetal rhombencephalic neurons.Interactive effect of alcohol and nicotine on developing cerebellum: an investigation of the temporal pattern of alcohol and nicotine administration.Increased tunel positive cells in CA1, CA2, and CA3 subfields of rat hippocampus due to copper and ethanol co-exposure.Antioxidant pretreatment does not ameliorate alcohol-induced Purkinje cell loss in the developing rat cerebellum.Ultrastructure of rat pup's Purkinje neurons whose mothers were exposed to ethanol during pregnancy and lactation.Zinc deficiency as a mediator of toxic effects of alcohol abuse.
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P2860
Zinc supplementation does not attenuate alcohol-induced cerebellar Purkinje cell loss during the brain growth spurt period.
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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name
Zinc supplementation does not ...... the brain growth spurt period.
@en
Zinc supplementation does not ...... the brain growth spurt period.
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type
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Zinc supplementation does not ...... the brain growth spurt period.
@en
Zinc supplementation does not ...... the brain growth spurt period.
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Zinc supplementation does not ...... the brain growth spurt period.
@en
Zinc supplementation does not ...... the brain growth spurt period.
@nl
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Zinc supplementation does not ...... the brain growth spurt period.
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10.1111/J.1530-0277.2001.TB02256.X
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2001-04-01T00:00:00Z