Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

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Nitrosative stress and transcription.Glutathione depletion is necessary for apoptosis in lymphoid cells independent of reactive oxygen species formation Role of nitric oxide, reactive oxygen species, and p38 MAP kinase in the regulation of human chondrocyte apoptosis.

P2860

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P2860

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

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http://www.wikidata.org/entity/Q43635660

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2001 nî lūn-bûn

@nan

2001年の論文

@ja

2001年学术文章

@wuu

2001年学术文章

@zh

2001年学术文章

@zh-cn

2001年学术文章

@zh-hans

2001年学术文章

@zh-my

2001年学术文章

@zh-sg

2001年學術文章

@yue

2001年學術文章

@zh-hant

name

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@en

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@nl

type

Item

label

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@en

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@nl

prefLabel

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@en

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

@nl

P1476

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis

@en

P2093

A Rossi

http://www.w3.org/2001/XMLSchema#string

F Bernassola

http://www.w3.org/2001/XMLSchema#string

G Melino

http://www.w3.org/2001/XMLSchema#string

M V Catani

http://www.w3.org/2001/XMLSchema#string

P2860

Human Fas ligand: gene structure, chromosomal location and species specificity

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

Nitric oxide inhibits tumor necrosis factor-alpha-induced apoptosis by reducing the generation of ceramide

Nitric oxide inhibits execution of apoptosis at two distinct ATP-dependent steps upstream and downstream of mitochondrial cytochrome c release

P304

123-133

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/JCB.1143

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Marco Corazzari

P577

2001-04-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

11400169

http://www.w3.org/2001/XMLSchema#string

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

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P2860

P2860

Inactivation of multiple targets by nitric oxide in CD95-triggered apoptosis.

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698