Growth retardation, duodenal lesions, and aberrant ileum architecture in triple null mice lacking EGF, amphiregulin, and TGF-alpha.
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Vanin-1(-/-) mice show decreased NSAID- and Schistosoma-induced intestinal inflammation associated with higher glutathione storesEpigen, the last ligand of ErbB receptors, reveals intricate relationships between affinity and mitogenicityCreating diseases to understand what prevents them: genetic analysis of inflammation in the gastrointestinal tractEpidermal growth factor, from gene organization to bedsideBetacellulin stimulates growth of the mouse intestinal epithelium and increases adenoma multiplicity in Apc+/Min miceAmphiregulin is a novel growth factor involved in normal bone development and in the cellular response to parathyroid hormone stimulationRegulation of heparin-binding EGF-like growth factor by miR-212 and acquired cetuximab-resistance in head and neck squamous cell carcinomaMyD88 signaling in nonhematopoietic cells protects mice against induced colitis by regulating specific EGF receptor ligandsThe epidermal growth factor receptor family: biology driving targeted therapeutics.Effect of breast milk and weaning on epithelial growth of the small intestine in humans.Colon-specific delivery of a probiotic-derived soluble protein ameliorates intestinal inflammation in mice through an EGFR-dependent mechanism.Defective valvulogenesis in HB-EGF and TACE-null mice is associated with aberrant BMP signaling.Human transforming growth factor alpha (TGF-alpha) is digested to a smaller (1-43), less biologically active, form in acidic gastric juiceLrig1: a new master regulator of epithelial stem cells.Enterocyte expression of epidermal growth factor receptor is not required for intestinal adaptation in response to massive small bowel resectionEGF and amphiregulin differentially regulate Cbl recruitment to endosomes and EGF receptor fate.Development of the infant intestine: implications for nutrition support.Reg4+ deep crypt secretory cells function as epithelial niche for Lgr5+ stem cells in colon.EGFR family: structure physiology signalling and therapeutic targets.ERBBs in the gastrointestinal tract: recent progress and new perspectives.Mitochondrial reactive oxygen species mediate GPCR-induced TACE/ADAM17-dependent transforming growth factor-alpha shedding.Epiregulin is not essential for development of intestinal tumors but is required for protection from intestinal damage.Identification of a cKit(+) colonic crypt base secretory cell that supports Lgr5(+) stem cells in mice.Epidermal growth factor signalling and bone metastasis.The EGFR-HER2 module: a stem cell approach to understanding a prime target and driver of solid tumors.The sorting protein PACS-2 promotes ErbB signalling by regulating recycling of the metalloproteinase ADAM17.Epidermal growth factor stimulates Rac activation through Src and phosphatidylinositol 3-kinase to promote colonic epithelial cell migration.EGFR signaling blunts allergen-induced IL-6 production and Th17 responses in the skin and attenuates development and relapse of atopic dermatitis.EGFR/Ras/MAPK signaling mediates adult midgut epithelial homeostasis and regeneration in Drosophila.Effects of growth factors and receptor blockade on gastrointestinal cancer.Role of epidermal growth factor and its receptor in chemotherapy-induced intestinal injury.Suppression of early growth response factor-1 with egr-1 antisense oligodeoxynucleotide aggravates experimental duodenal ulcers.Loss of PACS-2 delays regeneration in DSS-induced colitis but does not affect the ApcMin model of colorectal cancer.Interplay between nuclear factor erythroid 2-related factor 2 and amphiregulin during mechanical ventilation.Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine.Neutrophils Promote Amphiregulin Production in Intestinal Epithelial Cells through TGF-β and Contribute to Intestinal HomeostasisTopical administration of EGF suppresses immune response and protects skin barrier in DNCB-induced atopic dermatitis in NC/Nga mice
P2860
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P2860
Growth retardation, duodenal lesions, and aberrant ileum architecture in triple null mice lacking EGF, amphiregulin, and TGF-alpha.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@en
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@nl
type
label
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@en
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@nl
prefLabel
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@en
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@nl
P2093
P356
P1433
P1476
Growth retardation, duodenal l ...... , amphiregulin, and TGF-alpha.
@en
P2093
P356
10.1053/GAST.2001.25478
P407
P50
P577
2001-07-01T00:00:00Z