Double gastric infection with Helicobacter pylori and non-Helicobacter pylori bacteria during acid-suppressive therapy: increase of pro-inflammatory cytokines and development of atrophic gastritis.
about
Long-term proton pump inhibitor (PPI) use and the development of gastric pre-malignant lesionsLong term proton pump inhibitor (PPI) use and incidence of gastric premalignant lesionsProbiotics against neoplastic transformation of gastric mucosa: effects on cell proliferation and polyamine metabolismUpper gastrointestinal microbiota and digestive diseasesDo we need gastric acid?Analysis of Gastric Body Microbiota by Pyrosequencing: Possible Role of Bacteria Other Than Helicobacter pylori in the Gastric Carcinogenesis.Bacterial flora concurrent with Helicobacter pylori in the stomach of patients with upper gastrointestinal diseases.Survival in hostile territory: the microbiota of the stomach.Reflux disease management strategy: initial therapy.H. pylori and its modulation of gastrointestinal microbiota.Analysis of Gastric Microbiota by Pyrosequencing: Minor Role of Bacteria Other Than Helicobacter pylori in the Gastric Carcinogenesis.Review article: the long-term use of proton-pump inhibitors.Comparison of Gastric Microbiota Between Gastric Juice and Mucosa by Next Generation Sequencing MethodPolymorphisms in Toll-like receptor genes and risk of cancer.Helicobacter pylori: host genetics and disease outcomes.Why is the coexistence of gastric cancer and duodenal ulcer rare? Examination of factors related to both gastric cancer and duodenal ulcer.Helicobacter pylori and other gastric bacteria.Premalignant conditions of gastric cancer.Safety of proton pump inhibitors and risk of gastric cancers: review of literature and pathophysiological mechanisms.Acid-Suppressive Therapy and Risk of Infections: Pros and Cons.Proton pump inhibitors and gastric neoplasia.Proton-pump inhibitors: understanding the complications and risks.Helicobacter pylori attenuates lipopolysaccharide-induced nitric oxide production by murine macrophages.Long-term acid suppression by omeprazole in gastro-oesophageal reflux disease patients does not lead to anti-gastric autoantibody production.Changes of gastric mucosal architecture during long-term omeprazole therapy: results of a randomized clinical trial.
P2860
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P2860
Double gastric infection with Helicobacter pylori and non-Helicobacter pylori bacteria during acid-suppressive therapy: increase of pro-inflammatory cytokines and development of atrophic gastritis.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Double gastric infection with ...... lopment of atrophic gastritis.
@en
Double gastric infection with ...... lopment of atrophic gastritis.
@nl
type
label
Double gastric infection with ...... lopment of atrophic gastritis.
@en
Double gastric infection with ...... lopment of atrophic gastritis.
@nl
prefLabel
Double gastric infection with ...... lopment of atrophic gastritis.
@en
Double gastric infection with ...... lopment of atrophic gastritis.
@nl
P2093
P2860
P1476
Double gastric infection with ...... lopment of atrophic gastritis.
@en
P2093
De Bruïne A
Hameeteman W
Sanduleanu S
Stockbrügger RW
P2860
P304
P356
10.1046/J.1365-2036.2001.01029.X
P407
P577
2001-08-01T00:00:00Z