BRAF mutation in sporadic colorectal cancer and Lynch syndrome. - Wikidata - wikimedia - TriplyDB

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

http://www.wikidata.org/entity/Q43688934

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Toward a Molecular Classification of Colorectal Cancer: The Role of BRAF BRAFV600E immunohistochemistry in conjunction with mismatch repair status predicts survival in patients with colorectal cancer.Detection of the BRAF V600E mutation in colon carcinoma: critical evaluation of the imunohistochemical approach A further investigation of combined mismatch repair and BRAFV600E mutation specific immunohistochemistry as a predictor of overall survival in colorectal carcinoma Immunohistochemistry with the anti-BRAF V600E (VE1) antibody: impact of pre-analytical conditions and concordance with DNA sequencing in colorectal and papillary thyroid carcinoma.Immunohistochemical detection of the BRAF V600E mutant protein in colorectal neoplasms Oncogenic BRAF(V600E) Induces Clastogenesis and UVB Hypersensitivity DNA hypermethylation appears early and shows increased frequency with dysplasia in Lynch syndrome-associated colorectal adenomas and carcinomas.Lack of BRAFV600E mutation in stage I and II of colorectal cancer.Combination of microsatellite instability and BRAF mutation status for subtyping colorectal cancer.MLH1 constitutional and somatic methylation in patients with MLH1 negative tumors fulfilling the revised Bethesda criteria.Colorectal serrated pathway cancers and precursors.Microsatellite instability: an update.The distinctive molecular, pathological and clinical characteristics of BRAF-mutant colorectal tumors.BRAF in metastatic colorectal cancer: the future starts now.Prognostic value of BRAF V600E mutation and microsatellite instability in Japanese patients with sporadic colorectal cancer.Prevalence of somatic mutl homolog 1 promoter hypermethylation in Lynch syndrome colorectal cancer.First description of mutational analysis of MLH1, MSH2 and MSH6 in Algerian families with suspected Lynch syndrome.Immunohistochemistry with Anti-BRAF V600E (VE1) Mouse Monoclonal Antibody is a Sensitive Method for Detection of the BRAF V600E Mutation in Colon Cancer: Evaluation of 120 Cases with and without KRAS Mutation and Literature Review.Clinical Aspects of Microsatellite Instability Testing in Colorectal Cancer.Utility of BRAF V600E mutation-specific immunohistochemistry in detecting BRAF V600E-mutated gastrointestinal stromal tumors.Immunohistochemical staining for p16 and BRAFV600E is useful to distinguish between sporadic and hereditary (Lynch syndrome-related) microsatellite instable colorectal carcinomas.Comprehensive screening for mutations associated with colorectal cancer in unselected cases reveals penetrant and nonpenetrant mutations.Genomic profile of pseudomyxoma peritonei analyzed using next-generation sequencing and immunohistochemistry

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BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

http://www.wikidata.org/entity/Q43688934

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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name

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@en

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@nl

type

label

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@en

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@nl

prefLabel

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@en

BRAF mutation in sporadic colorectal cancer and Lynch syndrome.

@nl

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Virchows Archiv

P1476

BRAF mutation in sporadic colorectal cancer and Lynch syndrome

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P2093

Alexandra Thiel

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Annette Gylling

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Ari Ristimäki

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Arto Orpana

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Jonas Kantonen

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Mari Korhonen

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Mira Heinonen

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Soili Kytölä

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P2860

EGAPP supplementary evidence review: DNA testing strategies aimed at reducing morbidity and mortality from Lynch syndrome

The clinical phenotype of Lynch syndrome due to germ-line PMS2 mutations

Detection of BRAF V600E mutation in colorectal cancer: comparison of automatic sequencing and real-time chemistry methodology

Massively parallel tumor multigene sequencing to evaluate response to panitumumab in a randomized phase III study of metastatic colorectal cancer.

Concomitant BRAF and PI3K/mTOR blockade is required for effective treatment of BRAF(V600E) colorectal cancer.

A genetic progression model of Braf(V600E)-induced intestinal tumorigenesis reveals targets for therapeutic intervention.

Assessment of BRAF V600E mutation status by immunohistochemistry with a mutation-specific monoclonal antibody

BRAF mutation is frequently present in sporadic colorectal cancer with methylated hMLH1, but not in hereditary nonpolyposis colorectal cancer

Immunohistochemical testing of BRAF V600E status in 1,120 tumor tissue samples of patients with brain metastases

Systematic review of microsatellite instability and colorectal cancer prognosis

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s00428-013-1470-9

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5

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463

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Päivi Peltomäki

Jukka-Pekka Mecklin

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2013-08-21T00:00:00Z

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256072098

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colorectal cancer