Evidence against high glucose as a mediator of ERK1/2 or p38 MAPK phosphorylation in rat skeletal muscle.
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Improved insulin sensitivity with calorie restriction does not require reduced JNK1/2, p38, or ERK1/2 phosphorylation in skeletal muscle of 9-month-old rats.Prior exercise increases basal and insulin-induced p38 mitogen-activated protein kinase phosphorylation in human skeletal muscle.NKCC activity restores muscle water during hyperosmotic challenge independent of insulin, ERK, and p38 MAPK.
P2860
Evidence against high glucose as a mediator of ERK1/2 or p38 MAPK phosphorylation in rat skeletal muscle.
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2001 nî lūn-bûn
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2001年の論文
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Evidence against high glucose ...... lation in rat skeletal muscle.
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Evidence against high glucose ...... lation in rat skeletal muscle.
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type
label
Evidence against high glucose ...... lation in rat skeletal muscle.
@en
Evidence against high glucose ...... lation in rat skeletal muscle.
@nl
prefLabel
Evidence against high glucose ...... lation in rat skeletal muscle.
@en
Evidence against high glucose ...... lation in rat skeletal muscle.
@nl
P2093
P2860
P1476
Evidence against high glucose ...... lation in rat skeletal muscle.
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P2093
Wallberg-Henriksson H
P2860
P304
P356
10.1152/AJPENDO.2001.281.6.E1255
P577
2001-12-01T00:00:00Z