MHCI promotes developmental synapse elimination and aging-related synapse loss at the vertebrate neuromuscular junction.

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Major Histocompatibility Complex I Expression by Motor Neurons and Its Implication in Amyotrophic Lateral Sclerosis DOK7 gene therapy enhances motor activity and life span in ALS model mice.Schwann cells participate in synapse elimination at the developing neuromuscular junction.

P2860

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MHCI promotes developmental synapse elimination and aging-related synapse loss at the vertebrate neuromuscular junction.

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http://www.wikidata.org/entity/Q43899926

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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P1476

MHCI promotes developmental sy ...... ebrate neuromuscular junction.

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P2093

Lisa M Boulanger

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Marin A McDonald

http://www.w3.org/2001/XMLSchema#string

Mazell M Tetruashvily

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P2860

Multiple autism-linked genes mediate synapse elimination via proteasomal degradation of a synaptic scaffold PSD-95

Quantal components of the end-plate potential

MHC class I modulates NMDA receptor function and AMPA receptor trafficking

Functional requirement for class I MHC in CNS development and plasticity

A MusD retrotransposon insertion in the mouse Slc6a5 gene causes alterations in neuromuscular junction maturation and behavioral phenotypes

β2-microglobulin is a systemic pro-aging factor that impairs cognitive function and neurogenesis

Development of the vertebrate neuromuscular junction

Insights into the ageing mind: a view from cognitive neuroscience

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

The classical complement cascade mediates CNS synapse elimination

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197-208

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scholarly article

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10.1016/J.BBI.2016.01.008

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Lisa M Boulanger

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2016-01-20T00:00:00Z

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26802986

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5813483

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MHCI promotes developmental synapse elimination and aging-related synapse loss at the vertebrate neuromuscular junction.

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P2860

P2860

MHCI promotes developmental synapse elimination and aging-related synapse loss at the vertebrate neuromuscular junction.

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P1433

P1476

P2093

P2860

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P698

P932