Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
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Acute neonatal glucocorticoid exposure produces selective and rapid cerebellar neural progenitor cell apoptotic deathNicotinamide protects against ethanol-induced apoptotic neurodegeneration in the developing mouse brain.Fetal Alcohol Spectrum Disorder: Potential Role of Endocannabinoids SignalingTwo-Photon Enzymatic Probes Visualizing Sub-cellular/Deep-brain Caspase Activities in Neurodegenerative ModelsEthanol-induced neuronal apoptosis in vivo requires BAX in the developing mouse brainNuclear clusterin is associated with neuronal apoptosis in the developing rat brain upon ethanol exposureCaspase-mediated cleavage of actin and tubulin is a common feature and sensitive marker of axonal degeneration in neural development and injuryPotential of ketamine and midazolam, individually or in combination, to induce apoptotic neurodegeneration in the infant mouse brain.Postnatal day 7 ethanol treatment causes persistent reductions in adult mouse brain volume and cortical neurons with sex specific effects on neurogenesis.The role of NOX enzymes in ethanol-induced oxidative stress and apoptosis in mouse embryos.Dysregulation of TrkB phosphorylation and proBDNF protein in adenylyl cyclase 1 and 8 knockout mice in a model of fetal alcohol spectrum disorderTau phosphorylation and cleavage in ethanol-induced neurodegeneration in the developing mouse brain.Long-term consequences of developmental alcohol exposure on brain structure and function: therapeutic benefits of physical activityWhat the Spectrum of Microglial Functions Can Teach us About Fetal Alcohol Spectrum Disorder.Adenylyl cylases 1 and 8 mediate select striatal-dependent behaviors and sensitivity to ethanol stimulation in the adolescent period following acute neonatal ethanol exposure.Extracellular ATP protects pancreatic duct epithelial cells from alcohol-induced damage through P2Y1 receptor-cAMP signal pathway.Critical period for sensory experience-dependent survival of newly generated granule cells in the adult mouse olfactory bulbMolecular pathways underpinning ethanol-induced neurodegeneration.Effect of folic acid in prenatal alcohol induced behavioral impairment in Swiss albino mice.Focus on apoptosis to decipher how alcohol and many other drugs disrupt brain development.Modulation of GABAergic and glutamatergic transmission by ethanol in the developing neocortex: an in vitro test of the excessive inhibition hypothesis of fetal alcohol spectrum disorder.Alcohol-induced neuroapoptosis in the fetal macaque brain.Nrf2-mediated transcriptional induction of antioxidant response in mouse embryos exposed to ethanol in vivo: implications for the prevention of fetal alcohol spectrum disordersAdenylyl cyclases types 1 and 8 promote pro-survival pathways after ethanol exposure in the neonatal brainNeuroprotection with metformin and thymoquinone against ethanol-induced apoptotic neurodegeneration in prenatal rat cortical neurons.Cdc42-dependent activation of NADPH oxidase is involved in ethanol-induced neuronal oxidative stress.Neuroprotection by taurine in ethanol-induced apoptosis in the developing cerebellumMinocycline exacerbates apoptotic neurodegeneration induced by the NMDA receptor antagonist MK-801 in the early postnatal mouse brain.Pre-treatment with a Xingnaojing preparation ameliorates sevoflurane-induced neuroapoptosis in the infant rat striatumOverexpression of glycogen synthase kinase 3beta sensitizes neuronal cells to ethanol toxicityDifferential effects of ethanol on bid, tBid, and Bax:tBid interactions in postnatal day 4 and postnatal day 7 rat cerebellum.Resveratrol restores Nrf2 level and prevents ethanol-induced toxic effects in the cerebellum of a rodent model of fetal alcohol spectrum disordersProtective effect of calpain inhibitor N-acetyl-L-leucyl-L-leucyl-L-norleucinal on acute alcohol consumption related cardiomyopathy.Local and regional network function in behaviorally relevant cortical circuits of adult mice following postnatal alcohol exposureDifferential effects of ethanol on c-jun N-terminal kinase, 14-3-3 proteins, and Bax in postnatal day 4 and postnatal day 7 rat cerebellum.The clinically available NMDA receptor antagonist, memantine, exhibits relative safety in the developing rat brain.Lithium prevents long-term neural and behavioral pathology induced by early alcohol exposureFetal alcohol syndrome at the cellular level.Deficient PKR in RAX/PKR Association Ameliorates Ethanol-Induced Neurotoxicity in the Developing CerebellumPropylene glycol produces excessive apoptosis in the developing mouse brain, alone and in combination with phenobarbital
P2860
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P2860
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@en
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@nl
type
label
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@en
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@nl
prefLabel
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@en
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@nl
P2093
P356
P1476
Ethanol-induced caspase-3 activation in the in vivo developing mouse brain.
@en
P2093
Cleta D'Sa
John W Olney
Krikor Dikranian
Louis J Muglia
Tatyana Tenkova
P304
P356
10.1006/NBDI.2001.0475
P577
2002-03-01T00:00:00Z