Chronic atomoxetine treatment during adolescence decreases impulsive choice, but not impulsive action, in adult rats and alters markers of synaptic plasticity in the orbitofrontal cortex.
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Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluationReducing substance use during adolescence: a translational framework for preventionGambling disorder: an integrative review of animal and human studies.Norepinephrine and impulsivity: effects of acute yohimbineSensitivity to cognitive effort mediates psychostimulant effects on a novel rodent cost/benefit decision-making taskCocaine-seeking behavior in a genetic model of attention-deficit/hyperactivity disorder following adolescent methylphenidate or atomoxetine treatmentsDo the adjusting-delay and increasing-delay tasks measure the same construct: delay discounting?Performance on an impulse control task is altered in adult rats exposed to amphetamine during adolescence.At the Fulcrum in Health and Disease: Cdk5 and the Balancing Acts of Neuronal Structure and Physiology.Atomoxetine reduces hyperactive/impulsive behaviours in neurokinin-1 receptor 'knockout' mice.Effects of the combination of wheel running and atomoxetine on cue- and cocaine-primed reinstatement in rats selected for high or low impulsivity.Disadvantageous decision-making on a rodent gambling task is associated with increased motor impulsivity in a population of male rats.Dissociable effects of monoamine reuptake inhibitors on distinct forms of impulsive behavior in rats.The behavioral economics of substance use disorders: reinforcement pathologies and their repair.Chronic corticosterone exposure during adolescence reduces impulsive action but increases impulsive choice and sensitivity to yohimbine in male Sprague-Dawley rats.Neurogenetics and Epigenetics in Impulsive Behaviour: Impact on Reward CircuitrySimultaneous blockade of dopamine and noradrenaline reuptake promotes disadvantageous decision making in a rat gambling task.Performance on a strategy set shifting task during adolescence in a genetic model of attention deficit/hyperactivity disorder: methylphenidate vs. atomoxetine treatmentsDevelopmentally divergent effects of Rho-kinase inhibition on cocaine- and BDNF-induced behavioral plasticity.Prefrontal cortical BDNF: A regulatory key in cocaine- and food-reinforced behaviors.Rats are the smart choice: Rationale for a renewed focus on rats in behavioral genetics.The ontogeny of learned inhibition.Biological contributions to addictions in adolescents and adults: prevention, treatment, and policy implications.Translatable and Back-Translatable Measurement of Impulsivity and Compulsivity: Convergent and Divergent Processes.Setting the occasion for adolescent inhibitory control.Neural substrates of impulsive decision making modulated by modafinil in alcohol-dependent patients.Preventative treatment in an animal model of ADHD: Behavioral and biochemical effects of methylphenidate and its interactions with ovarian hormones in female rats.Noradrenergic modulation of risk/reward decision making.Adolescent atomoxetine treatment in a rodent model of ADHD: effects on cocaine self-administration and dopamine transporters in frontostriatal regions.Sex differences in the reduction of impulsive choice (delay discounting) for cocaine in rats with atomoxetine and progesterone.Subchronic administration of atomoxetine causes an enduring reduction in context-induced relapse to cocaine seeking without affecting impulsive decision making.Increased cocaine-induced conditioned place preference during periadolescence in maternally separated male BALB/c mice: the role of cortical BDNF, microRNA-212, and MeCP2.Dissecting drug effects in preclinical models of impulsive choice: emphasis on glutamatergic compounds.Right prefrontal and ventral striatum interactions underlying impulsive choice and impulsive responding.Changes in the serum levels of brain-derived neurotrophic factor in adults with attention deficit hyperactivity disorder after treatment with atomoxetine.Atomoxetine accelerates attentional set shifting without affecting learning rate in the rat.Methylphenidate and Atomoxetine-Responsive Prefrontal Cortical Genetic Overlaps in "Impulsive" SHR/NCrl and Wistar Rats.
P2860
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P2860
Chronic atomoxetine treatment during adolescence decreases impulsive choice, but not impulsive action, in adult rats and alters markers of synaptic plasticity in the orbitofrontal cortex.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@en
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@nl
type
label
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@en
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@nl
prefLabel
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@en
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@nl
P2093
P2860
P1433
P1476
Chronic atomoxetine treatment ...... y in the orbitofrontal cortex.
@en
P2093
Catharine A Winstanley
Fiona D Zeeb
Haosheng Sun
P2860
P2888
P304
P356
10.1007/S00213-011-2419-9
P577
2011-08-02T00:00:00Z
P5875
P6179
1026052533