Retinal organization in the bcl-2-overexpressing transgenic mouse.
about
Low-level gestational lead exposure increases retinal progenitor cell proliferation and rod photoreceptor and bipolar cell neurogenesis in miceGestational lead exposure selectively decreases retinal dopamine amacrine cells and dopamine content in adult miceComplexity of retinal cone bipolar cellsMajor role of BAX in apoptosis during retinal development and in establishment of a functional postnatal retinaGenomic control of neuronal demographics in the retinaMultiple genes on chromosome 7 regulate dopaminergic amacrine cell number in the mouse retina.The composition of the inner nuclear layer of the cat retina.Programmed cell death of retinal cone bipolar cells is independent of afferent or target controlDevelopment and degeneration of cone bipolar cells are independent of cone photoreceptors in a mouse model of retinitis pigmentosa.Design principles and developmental mechanisms underlying retinal mosaics.Analysis of bipolar and amacrine populations in marmoset retina.Defining the role of the Bcl-2 family of proteins in the nervous system.Optomotor and immunohistochemical changes in the juvenile S334ter ratProlonged expression of Puma in cholinergic amacrine cells during the development of rat retina.Spatial patterning of cholinergic amacrine cells in the mouse retinaGenomic Control of Retinal Cell Number: Challenges, Protocol, and Results.Morphology of dopaminergic amacrine cells in the mouse retina: independence from homotypic interactions.Integrative properties of retinal ganglion cell electrical responsiveness depend on neurotrophic support and genotype in the mouse.Light, lipids and photoreceptor survival: live or let die?BBC3 (PUMA) regulates developmental apoptosis but not axonal injury induced death in the retina.The mouse pattern electroretinogram.Botulinum neurotoxin A impairs neurotransmission following retrograde transynaptic transport.Cellular positioning and dendritic field size of cholinergic amacrine cells are impervious to early ablation of neighboring cells in the mouse retina.Regularity and packing of the horizontal cell mosaic in different strains of mice.Developmental improvement in the regularity and packing of mouse horizontal cells: implications for mechanisms underlying mosaic pattern formation.Prdm13 is required for Ebf3+ amacrine cell formation in the retina.Brn3a and Brn3b knockout mice display unvaried retinal fine structure despite major morphological and numerical alterations of ganglion cells.Cyanidin-3-glucoside and its phenolic acid metabolites attenuate visible light-induced retinal degeneration in vivo via activation of Nrf2/HO-1 pathway and NF-κB suppression.
P2860
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P2860
Retinal organization in the bcl-2-overexpressing transgenic mouse.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@en
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@nl
type
label
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@en
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@nl
prefLabel
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@en
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@nl
P2860
P356
P1476
Retinal organization in the bcl-2-overexpressing transgenic mouse.
@en
P2093
Maila Volpini
P2860
P356
10.1002/CNE.10177
P407
P577
2002-04-01T00:00:00Z