Oxidative stress activation of miR-125b is part of the molecular switch for Hailey-Hailey disease manifestation.
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Nitric oxide, oxidative stress, and p66Shc interplay in diabetic endothelial dysfunctionSubstrate-dependent gene regulation of self-assembled human MSC spheroids on chitosan membranes.Loss of Notch1-dependent p21(Waf1/Cip1) expression influences the Notch1 outcome in tumorigenesis.Efficacy of the melanocortin analogue Nle4-D-Phe7-α-melanocyte-stimulating hormone in the treatment of patients with Hailey-Hailey disease.The role of microRNAs in the biology of rare diseases.Hailey-Hailey disease improved by fractional CO2 laser.H-ferritin-regulated microRNAs modulate gene expression in K562 cellsTopical hesperidin improves epidermal permeability barrier function and epidermal differentiation in normal murine skin.LncRNAs H19 and HULC, activated by oxidative stress, promote cell migration and invasion in cholangiocarcinoma through a ceRNA mannerMicroRNAs play a central role in molecular dysfunctions linking inflammation with cancer.Oxidative stress and microRNAs in vascular diseases.How the TP53 family proteins TP63 and TP73 contribute to tumorigenesis: regulators and effectors.Pharmacokinetics and Pharmacodynamics of Afamelanotide and its Clinical Use in Treating Dermatologic Disorders.The Impact of Environmental Factors in Influencing Epigenetics Related to Oxidative States in the Cardiovascular System.Hailey-Hailey disease and tight junctions: Claudins 1 and 4 are regulated by ATP2C1 gene encoding Ca(2+) /Mn(2+) ATPase SPCA1 in cultured keratinocytes.The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease.Predicted overlapping microRNA regulators of acetylcholine packaging and degradation in neuroinflammation-related disorders.The deregulated expression of miR-125b in acute myeloid leukemia is dependent on the transcription factor C/EBPα.Overlapping ATP2C1 and ASTE1 genes in human genome: implications for SPCA1 expression?Nucleic acid-based non-invasive prenatal diagnosis of genetic skin diseases: are we ready?Association of prohibitin-1 and 2 with oxidative stress in rats with renal interstitial fibrosis.The IL-6/STAT3 pathway upregulates microRNA-125b expression in hepatitis C virus infection.Yeast-Based Screen to Identify Natural Compounds with a Potential Therapeutic Effect in Hailey-Hailey Disease.Predictive medicine and biomarkers: the case of rare diseases
P2860
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P2860
Oxidative stress activation of miR-125b is part of the molecular switch for Hailey-Hailey disease manifestation.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
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2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Oxidative stress activation of ...... -Hailey disease manifestation.
@en
Oxidative stress activation of ...... -Hailey disease manifestation.
@nl
type
label
Oxidative stress activation of ...... -Hailey disease manifestation.
@en
Oxidative stress activation of ...... -Hailey disease manifestation.
@nl
prefLabel
Oxidative stress activation of ...... -Hailey disease manifestation.
@en
Oxidative stress activation of ...... -Hailey disease manifestation.
@nl
P2093
P2860
P50
P1476
Oxidative stress activation of ...... -Hailey disease manifestation.
@en
P2093
Claudio Palleschi
Claudio Talora
Eleonora Candi
Gerry Melino
Gianfranco Biolcati
Isabella Screpanti
Karine Lefort
Marco Salvatore
Samantha Cialfi
P2860
P304
P356
10.1111/J.1600-0625.2011.01359.X
P577
2011-09-14T00:00:00Z