The conversion of redox status of peritoneal macrophages during pathological progression of spontaneous inflammatory bowel disease in Janus family tyrosine kinase 3(-/-) and IL-2 receptor gamma(-/-) mice.
about
Molecular mechanism of interleukin-2-induced mucosal homeostasisIntegrating Immunologic Signaling Networks: The JAK/STAT Pathway in Colitis and Colitis-Associated CancerRole of regulatory T cell in the pathogenesis of inflammatory bowel diseaseGenetically engineered mouse models for studying inflammatory bowel diseaseRole of Janus Kinase 3 in Predisposition to Obesity-associated Metabolic SyndromeUncovering Pathogenic Mechanisms of Inflammatory Bowel Disease Using Mouse Models of Crohn's Disease-Like Ileitis: What is the Right Model?Role of Janus kinase 3 in mucosal differentiation and predisposition to colitisJanus kinase 3 regulates interleukin 2-induced mucosal wound repair through tyrosine phosphorylation of villin.Potential molecular mechanism for c-Src kinase-mediated regulation of intestinal cell migration.Type 2 innate immune responses and the natural helper cell.Th2-type innate immune responses mediated by natural helper cells.miR-27b*, an oxidative stress-responsive microRNA modulates nuclear factor-kB pathway in RAW 264.7 cells.Chronic glycine treatment inhibits ligature-induced periodontal disease in Wistar rats.Glutathione redox regulates airway hyperresponsiveness and airway inflammation in mice.Multidose streptozotocin induction of diabetes in BALB/c mice induces a dominant oxidative macrophage and a conversion of TH1 to TH2 phenotypes during disease progressionThe thiol redox state of lymphoid organs is modified by immunization: role of different immune cell populations.Identification of inflammatory bowel disease-related proteins using a reverse k-nearest neighbor search.Hydrogen peroxide increases interleukin-12 p40/p70 molecular ratio and induces Th2-predominant responses in mice.
P2860
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P2860
The conversion of redox status of peritoneal macrophages during pathological progression of spontaneous inflammatory bowel disease in Janus family tyrosine kinase 3(-/-) and IL-2 receptor gamma(-/-) mice.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
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2002年學術文章
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name
The conversion of redox status ...... Janus family tyrosine kinase 3
@nl
The conversion of redox status ...... IL-2 receptor gamma(-/-) mice.
@en
type
label
The conversion of redox status ...... Janus family tyrosine kinase 3
@nl
The conversion of redox status ...... IL-2 receptor gamma(-/-) mice.
@en
prefLabel
The conversion of redox status ...... Janus family tyrosine kinase 3
@nl
The conversion of redox status ...... IL-2 receptor gamma(-/-) mice.
@en
P2093
P2860
P356
P1476
The conversion of redox status ...... IL-2 receptor gamma(-/-) mice.
@en
P2093
Akira Yamashita
Junji Hamuro
Kazuo Sugamura
Takashi Saito
Yukie Murata
P2860
P304
P356
10.1093/INTIMM/DXF031
P577
2002-06-01T00:00:00Z