Relations between brain pathology and temporal lobe epilepsy. - Wikidata - wikimedia - TriplyDB

Relations between brain pathology and temporal lobe epilepsy.

Relations between brain pathology and temporal lobe epilepsy.

http://www.wikidata.org/entity/Q44067016

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Cannabinoids promote embryonic and adult hippocampus neurogenesis and produce anxiolytic- and antidepressant-like effects Classic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single “cryptic” episode of focal hippocampal excitation in awake rats Epileptogenesis provoked by prolonged experimental febrile seizures: mechanisms and biomarkers.Gene expression changes after seizure preconditioning in the three major hippocampal cell layers.Does pilocarpine-induced epilepsy in adult rats require status epilepticus?Optimization of multiplexed bead-based cytokine immunoassays for rat serum and brain tissue Mechanisms explaining transitions between tonic and phasic firing in neuronal populations as predicted by a low dimensional firing rate model Neuroprotection by glutamate receptor antagonists against seizure-induced excitotoxic cell death in the aging brain.Tall gastrodis tuber combined with antiepileptic drugs repairs abnormal perfusion foci in focal epilepsy A chronic histopathological and electrophysiological analysis of a rodent hypoxic-ischemic brain injury model and its use as a model of epilepsy.Administration of simvastatin after kainic acid-induced status epilepticus restrains chronic temporal lobe epilepsy.Functional integration of new neurons into hippocampal networks and poststroke comorbidities following neonatal stroke in mice.Increased excitatory synaptic input to granule cells from hilar and CA3 regions in a rat model of temporal lobe epilepsy Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.Post-traumatic seizure susceptibility is attenuated by hypothermia therapy.Rapamycin suppresses mossy fiber sprouting but not seizure frequency in a mouse model of temporal lobe epilepsy.Epileptogenesis after prolonged febrile seizures: mechanisms, biomarkers and therapeutic opportunities.Promise of resveratrol for easing status epilepticus and epilepsy.Neuroprotective Effect of Uncaria rhynchophylla in Kainic Acid-Induced Epileptic Seizures by Modulating Hippocampal Mossy Fiber Sprouting, Neuron Survival, Astrocyte Proliferation, and S100B Expression.Combined role of seizure-induced dendritic morphology alterations and spine loss in newborn granule cells with mossy fiber sprouting on the hyperexcitability of a computer model of the dentate gyrus Age-dependent changes in intrinsic neuronal excitability in subiculum after status epilepticus.Plasticity of neuropeptide Y in the dentate gyrus after seizures, and its relevance to seizure-induced neurogenesis Neuropeptide Y in the recurrent mossy fiber pathway.The pilocarpine model of temporal lobe epilepsy The kainic acid model of temporal lobe epilepsy Bim regulation may determine hippocampal vulnerability after injurious seizures and in temporal lobe epilepsy.Enhanced tonic GABA current in normotopic and hilar ectopic dentate granule cells after pilocarpine-induced status epilepticus Morphological changes among hippocampal dentate granule cells exposed to early kindling-epileptogenesis.Morphologic integration of hilar ectopic granule cells into dentate gyrus circuitry in the pilocarpine model of temporal lobe epilepsy.Pathophysiogenesis of mesial temporal lobe epilepsy: is prevention of damage antiepileptogenic?Animal models of temporal lobe epilepsy following systemic chemoconvulsant administration Is mesial temporal sclerosis a necessary component of temporal lobe epilepsy?Proepileptic influence of a focal vascular lesion affecting entorhinal cortex-CA3 connections after status epilepticus Mossy fibers are the primary source of afferent input to ectopic granule cells that are born after pilocarpine-induced seizures.Resveratrol for Easing Status Epilepticus Induced Brain Injury, Inflammation, Epileptogenesis, and Cognitive and Memory Dysfunction-Are We There Yet?Implication of the Kallikrein-Kinin system in neurological disorders: Quest for potential biomarkers and mechanisms.The Free Radical Scavenger N-Tert-Butyl-α-Phenylnitrone (PBN) Administered to Immature Rats During Status Epilepticus Alters Neurogenesis and Has Variable Effects, Both Beneficial and Detrimental, on Long-Term Outcomes Targeting the Mouse Ventral Hippocampus in the Intrahippocampal Kainic Acid Model of Temporal Lobe Epilepsy

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P2860

Relations between brain pathology and temporal lobe epilepsy.

http://www.wikidata.org/entity/Q44067016

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

@zh-hans

2002年学术文章

@zh-my

2002年学术文章

@zh-sg

2002年學術文章

@yue

2002年學術文章

@zh

2002年學術文章

@zh-hant

name

Relations between brain pathology and temporal lobe epilepsy.

@en

Relations between brain pathology and temporal lobe epilepsy.

@nl

type

label

Relations between brain pathology and temporal lobe epilepsy.

@en

Relations between brain pathology and temporal lobe epilepsy.

@nl

prefLabel

Relations between brain pathology and temporal lobe epilepsy.

@en

Relations between brain pathology and temporal lobe epilepsy.

@nl

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JNEUROSCI.22-14-06052.2002

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Journal of Neuroscience

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Relations between brain pathology and temporal lobe epilepsy

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Amy E Wallace

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Darren K Hannesson

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Deborah M Saucier

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Larry C Schmued

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Michael E Corcoran

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Shu-Sen Cui

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Xia Zhang

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6052-6061

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scholarly article

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10.1523/JNEUROSCI.22-14-06052.2002

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William G. Honer

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2002-07-01T00:00:00Z

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12122066

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