Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats.
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Improved energy supply regulation in chronic hypoxic mouse counteracts hypoxia-induced altered cardiac energeticsChronic intermittent hypoxia alters Ca2+ handling in rat cardiomyocytes by augmented Na+/Ca2+ exchange and ryanodine receptor activities in ischemia-reperfusionRole of oxidative stress in PKC-delta upregulation and cardioprotection induced by chronic intermittent hypoxiaDiversity of mitochondria-dependent dilator mechanisms in vascular smooth muscle of cerebral arteries from normal and insulin-resistant rats.Mitochondrial energy metabolism plays a critical role in the cardioprotection afforded by intermittent hypobaric hypoxia.Mitochondrial BKCa channels contribute to protection of cardiomyocytes isolated from chronically hypoxic rats.Hypoxia. 4. Hypoxia and ion channel functionTherapeutic potential of intermittent hypoxia: a matter of dose.Cardiac response to chronic intermittent hypoxia with a transition from adaptation to maladaptation: the role of hydrogen peroxide.Depolarization of mitochondria in endothelial cells promotes cerebral artery vasodilation by activation of nitric oxide synthaseChronic intermittent hypoxia affects the cytosolic phospholipase A2α/cyclooxygenase 2 pathway via β2-adrenoceptor-mediated ERK/p38 stimulation.Mitochondria as a target of cardioprotection in models of preconditioning.Effect of sustained hypobaric hypoxia during maturation and aging on rat myocardium. I. Mechanical activity.Intermittent hypobaric hypoxia applicability in myocardial infarction prevention and recovery.Mitochondrial KATP opening confers protection against lethal myocardial injury and ischaemia-induced arrhythmias in the rat heart via PI3K/Akt-dependent and -independent mechanisms.Increased expression and altered subcellular distribution of PKC-delta in chronically hypoxic rat myocardium: involvement in cardioprotection.Chronic intermittent hypoxia increases infarction in the isolated rat heart.Inducible nitric oxide synthase contributes to intermittent hypoxia against ischemia/reperfusion injury.Antioxidant tempol suppresses heart cytosolic phospholipase A2α stimulated by chronic intermittent hypoxia.Cardioprotective adaptation of rats to intermittent hypobaric hypoxia is accompanied by the increased association of hexokinase with mitochondria.Cardioprotective and nonprotective regimens of chronic hypoxia diversely affect the myocardial antioxidant systems.Modulation of mitochondrial respiratory function and ROS production by novel benzopyran analogues.The mitochondrial K(ATP) channel opener BMS-191095 reduces neuronal damage after transient focal cerebral ischemia in rats.Cardioprotection by Intermittent Hypoxia Conditioning: Evidence, Mechanisms and Therapeutic Potential.Activation of α1B-adrenoceptors contributes to intermittent hypobaric hypoxia-improved postischemic myocardial performance via inhibiting MMP-2 activation.
P2860
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P2860
Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Effects of mitochondrial K
@nl
Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.
@en
type
label
Effects of mitochondrial K
@nl
Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.
@en
prefLabel
Effects of mitochondrial K
@nl
Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.
@en
P2093
P1476
Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.
@en
P2093
Bohuslav Ost'ádal
Frantisek Kolár
Frantisek Papousek
Gary J Grover
Jan Neckár
Lukás Koten
Ondrej Szárszoi
P304
P356
10.1016/S0008-6363(02)00456-X
P577
2002-08-01T00:00:00Z