Effect of indoleamine 2,3-dioxygenase on induction of experimental autoimmune encephalomyelitis.
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Kynurenines in CNS disease: regulation by inflammatory cytokinesCurrent Evidence for a Role of the Kynurenine Pathway of Tryptophan Metabolism in Multiple SclerosisIndoleamine 2,3 Dioxygenase as a Potential Therapeutic Target in Huntington's DiseaseThe Role of Indoleamine 2, 3-Dioxygenase in Immune Suppression and Autoimmunity1-Methyl-tryptophan can interfere with TLR signaling in dendritic cells independently of IDO activityIDO2: A Pathogenic Mediator of Inflammatory AutoimmunityExpression analysis of novel striatal-enriched genes in Huntington disease.Expression of indoleamine 2,3-dioxygenase in nasopharyngeal carcinoma impairs the cytolytic function of peripheral blood lymphocytes.Decreased IDO activity and increased TTS expression break immune tolerance in patients with immune thrombocytopenia.The chemokine receptor antagonist, TAK-779, decreased experimental autoimmune encephalomyelitis by reducing inflammatory cell migration into the central nervous system, without affecting T cell function.Regulatory T cells in multiple sclerosis and myasthenia gravis.Activation of the STING adaptor attenuates experimental autoimmune encephalitis.Dendritic cell-regulatory T-cell interactions control self-directed immunity.GM-CSF-neuroantigen fusion proteins reverse experimental autoimmune encephalomyelitis and mediate tolerogenic activity in adjuvant-primed environments: association with inflammation-dependent, inhibitory antigen presentation.IDO upregulates regulatory T cells via tryptophan catabolite and suppresses encephalitogenic T cell responses in experimental autoimmune encephalomyelitisIDO2 is a critical mediator of autoantibody production and inflammatory pathogenesis in a mouse model of autoimmune arthritis.Regulation of Th1/Th17 cytokines and IDO gene expression by inhibition of calpain in PBMCs from MS patients.Lipopolysaccharide-induced depressive-like behavior is mediated by indoleamine 2,3-dioxygenase activation in mice.Quinolinic acid toxicity on oligodendroglial cells: relevance for multiple sclerosis and therapeutic strategies.Effect of immunoglobin-like transcript 7 cross-linking on plasmacytoid dendritic cells differentiation into antigen-presenting cells.Indoleamine 2,3-dioxygenase, tryptophan catabolism, and Mycobacterium avium subsp. paratuberculosis: a model for chronic mycobacterial infections.Myeloid differentiation primary response protein 88 blockade upregulates indoleamine 2,3-dioxygenase expression in rheumatoid synovial fibroblasts.Amino acid catabolism: a pivotal regulator of innate and adaptive immunity.Inhibition of indoleamine 2,3-dioxygenase promotes vascular inflammation and increases atherosclerosis in Apoe-/- mice.Suppression of humoral immune response to hepatitis B surface antigen vaccine in BALB/c mice by 1-methyl-tryptophan co-administration.Indoleamine 2,3 Dioxygenase (IDO) Expression and Activity in Relapsing-Remitting Multiple Sclerosis.HIV inhibits CD4+ T-cell proliferation by inducing indoleamine 2,3-dioxygenase in plasmacytoid dendritic cellsIndoleamine 2,3-Dioxygenase Is Involved in the Inflammation Response of Corneal Epithelial Cells to Aspergillus fumigatus InfectionsExpression of the Kynurenine Pathway in Human Peripheral Blood Mononuclear Cells: Implications for Inflammatory and Neurodegenerative DiseaseExpression of indoleamine 2,3-dioxygenase by plasmacytoid dendritic cells in tumor-draining lymph nodes.Inhibition of indoleamine 2,3-dioxygenase (IDO) enhances elimination of virus-infected macrophages in an animal model of HIV-1 encephalitisIDO1 Deficiency Does Not Affect Disease in Mouse Models of Systemic Juvenile Idiopathic Arthritis and Secondary Hemophagocytic Lymphohistiocytosis.Addition of an indoleamine 2,3,-dioxygenase inhibitor to B cell-depletion therapy blocks autoreactive B cell activation and recurrence of arthritis in K/BxN mice.Anti-inflammatory mechanisms of IFN-γ studied in experimental autoimmune encephalomyelitis reveal neutrophils as a potential target in multiple sclerosis.Mouse mesenchymal stem cells suppress antigen-specific TH cell immunity independent of indoleamine 2,3-dioxygenase 1 (IDO1).Identification of Interferon-Stimulated Genes with Antiretroviral Activity.3-Hydroxyanthranilic acid inhibits PDK1 activation and suppresses experimental asthma by inducing T cell apoptosis.Allosteric modulation of metabotropic glutamate receptor 4 activates IDO1-dependent, immunoregulatory signaling in dendritic cells.IFN-gamma-induced IDO and WRS expression in microglia is differentially regulated by IL-4.Dendritic cells, indoleamine 2,3 dioxygenase and acquired immune privilege
P2860
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P2860
Effect of indoleamine 2,3-dioxygenase on induction of experimental autoimmune encephalomyelitis.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@en
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@nl
type
label
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@en
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@nl
prefLabel
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@en
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@nl
P2093
P1476
Effect of indoleamine 2,3-diox ...... autoimmune encephalomyelitis.
@en
P2093
Gene M Shearer
Jerrold M Ward
Jian-Ping Zou
Jolynne R Tschetter
Kenichi Sakurai
P304
P356
10.1016/S0165-5728(02)00176-5
P577
2002-08-01T00:00:00Z