Phenylethyl isothiocyanate induces apoptotic signaling via suppressing phosphatase activity against c-Jun N-terminal kinase.
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Molecular targets of dietary phenethyl isothiocyanate and sulforaphane for cancer chemopreventionMitogen Activated Protein kinase signal transduction pathways in the prostateBioavailability of Glucosinolates and Their Breakdown Products: Impact of ProcessingBasic principles and emerging concepts in the redox control of transcription factorsSulforaphane potentiates RNA damage induced by different xenobioticsNutrient isothiocyanates covalently modify and inhibit the inflammatory cytokine macrophage migration inhibitory factor (MIF)The isothiocyanate class of bioactive nutrients covalently inhibit the MEKK1 protein kinase.Molecular mechanisms underlying cochlear degeneration in the tubby mouse and the therapeutic effect of sulforaphane.Phenethyl isothiocyanate sensitizes androgen-independent human prostate cancer cells to docetaxel-induced apoptosis in vitro and in vivo.p66Shc is indispensable for phenethyl isothiocyanate-induced apoptosis in human prostate cancer cellsPhenethyl isothiocyanate inhibits oxidative phosphorylation to trigger reactive oxygen species-mediated death of human prostate cancer cellsDifferential response of normal (PrEC) and cancerous human prostate cells (PC-3) to phenethyl isothiocyanate-mediated changes in expression of antioxidant defense genes.Vaccinia H1-related phosphatase is a phosphatase of ErbB receptors and is down-regulated in non-small cell lung cancerChemopreventative potential of the cruciferous vegetable constituent phenethyl isothiocyanate in a mouse model of prostate cancer.Proteomic identification of binding targets of isothiocyanates: A perspective on techniques.Anti-tumor activity and signaling events triggered by the isothiocyanates, sulforaphane and phenethyl isothiocyanate, in multiple myelomaProteins as binding targets of isothiocyanates in cancer prevention.The molecular basis that unifies the metabolism, cellular uptake and chemopreventive activities of dietary isothiocyanates.Murine prostate cancer inhibition by dietary phytochemicals--curcumin and phenyethylisothiocyanateInhibition of androgen-responsive LNCaP prostate cancer cell tumor xenograft growth by dietary phenethyl isothiocyanate correlates with decreased angiogenesis and inhibition of cell attachment.Dietary phytochemicals and cancer prevention: Nrf2 signaling, epigenetics, and cell death mechanisms in blocking cancer initiation and progression.Isothiocyanates repress estrogen receptor alpha expression in breast cancer cellsAtg5 regulates phenethyl isothiocyanate-induced autophagic and apoptotic cell death in human prostate cancer cellsMitochondrial Dysfunction in Gliomas: Pharmacotherapeutic Potential of Natural Compounds.Anti-NF-kappaB and anti-inflammatory activities of synthetic isothiocyanates: effect of chemical structures and cellular signalingElectrophiles in foods: the current status of isothiocyanates and their chemical biology.MAP Kinases and Prostate Cancer.Phenethyl isothiocyanate alters the gene expression and the levels of protein associated with cell cycle regulation in human glioblastoma GBM 8401 cells.Phenethyl isothiocyanate promotes immune responses in normal BALB/c mice, inhibits murine leukemia WEHI-3 cells, and stimulates immunomodulations in vivo.Effects of phenylethyl isothiocyanate and its metabolite on cell-cycle arrest and apoptosis in LNCaP human prostate cancer cells.Activation of NF-E2-related factor-2 reverses biochemical dysfunction of endothelial cells induced by hyperglycemia linked to vascular disease.Biochemical and biological characterization of a neuroendocrine-associated phosphatase.Suppression of NF-kappaB and NF-kappaB-regulated gene expression by sulforaphane and PEITC through IkappaBalpha, IKK pathway in human prostate cancer PC-3 cells.Distinctive activation patterns in constitutively active and gefitinib-sensitive EGFR mutants.Measuring p66Shc Signaling Pathway Activation and Mitochondrial Translocation in Cultured Cells.ERK and JNK signaling pathways are involved in the regulation of activator protein 1 and cell death elicited by three isothiocyanates in human prostate cancer PC-3 cells.Inhibition of EGFR signaling in human prostate cancer PC-3 cells by combination treatment with beta-phenylethyl isothiocyanate and curcumin.Inhibition of Glycolysis in Prostate Cancer Chemoprevention by Phenethyl Isothiocyanate.
P2860
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P2860
Phenylethyl isothiocyanate induces apoptotic signaling via suppressing phosphatase activity against c-Jun N-terminal kinase.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@en
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@nl
type
label
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@en
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@nl
prefLabel
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@en
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@nl
P2093
P2860
P356
P1476
Phenylethyl isothiocyanate ind ...... ainst c-Jun N-terminal kinase.
@en
P2093
A-N Tony Kong
Rajashree Kori
Tse-Hua Tan
Yi-Rong Chen
P2860
P304
39334-39342
P356
10.1074/JBC.M202070200
P407
P577
2002-08-08T00:00:00Z