Monocyte chemotactic protein-1 directly induces human vascular smooth muscle proliferation.
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Bindarit inhibits human coronary artery smooth muscle cell proliferation, migration and phenotypic switchingEndogenous VEGF-A is responsible for mitogenic effects of MCP-1 on vascular smooth muscle cellsReactive oxygen species and ERK 1/2 mediate monocyte chemotactic protein-1-stimulated smooth muscle cell migrationDual delivery of VEGF and MCP-1 to support endothelial cell transplantation for therapeutic vascularizationHuman umbilical cord blood endothelial progenitor cells decrease vein graft neointimal hyperplasia in SCID mice.Collateralization and the response to obstruction of epicardial coronary arteries.Long-acting progestin-only contraceptives impair endometrial vasculature by inhibiting uterine vascular smooth muscle cell survival.Overexpression of mimecan in human aortic smooth muscle cells inhibits cell proliferation and enhances apoptosis and migration.The Features of Inflammation Factors Concentrations in Aqueous Humor of Polypoidal Choroidal VasculopathyMonocyte chemoattractant protein-1/CCR2 axis promotes vein graft neointimal hyperplasia through its signaling in graft-extrinsic cell populations.Myocardin inhibits cellular proliferation by inhibiting NF-kappaB(p65)-dependent cell cycle progression.Nf1+/- monocytes/macrophages induce neointima formation via CCR2 activation.The chemokine system in arteriogenesis and hind limb ischemia.The anti-inflammatory agent bindarit inhibits neointima formation in both rats and hyperlipidaemic mice.Prostacyclin inhibits IFN-gamma-stimulated cytokine expression by reduced recruitment of CBP/p300 to STAT1 in a SOCS-1-independent manner.Chlamydophila pneumoniae infection leads to smooth muscle cell proliferation and thickening in the coronary artery without contributions from a host immune response.Perivascular mast cells regulate vein graft neointimal formation and remodeling.Transplanted perivascular adipose tissue accelerates injury-induced neointimal hyperplasia: role of monocyte chemoattractant protein-1.Effects of tongxinluo on the neointima formation and expression of inflammatory cytokines in rats after carotid artery balloon injury.Airway epithelium stimulates smooth muscle proliferation.Abscisic acid released by human monocytes activates monocytes and vascular smooth muscle cell responses involved in atherogenesisβ-Amyloid context intensifies vascular smooth muscle cells induced inflammatory response and de-differentiation.Metformin-induced activation of AMPK inhibits the proliferation and migration of human aortic smooth muscle cells through upregulation of p53 and IFI16.AKT-independent activation of p38 MAP kinase promotes vascular calcification.
P2860
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P2860
Monocyte chemotactic protein-1 directly induces human vascular smooth muscle proliferation.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@en
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@nl
type
label
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@en
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@nl
prefLabel
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@en
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@nl
P2093
P1476
Monocyte chemotactic protein-1 ...... r smooth muscle proliferation.
@en
P2093
Alden H Harken
Craig H Selzman
Fabia Gamboni-Robertson
Michael A Zimmerman
Stephanie A Miller
P304
P356
10.1152/AJPHEART.00188.2002
P577
2002-06-20T00:00:00Z