Indolic uremic solutes increase tissue factor production in endothelial cells by the aryl hydrocarbon receptor pathway.
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Indoxyl Sulfate-Review of Toxicity and Therapeutic StrategiesThe aryl hydrocarbon receptor-activating effect of uremic toxins from tryptophan metabolism: a new concept to understand cardiovascular complications of chronic kidney diseaseElucidating mechanisms of toxicity using phenotypic data from primary human cell systems--a chemical biology approach for thrombosis-related side effects.Nutrients Turned into Toxins: Microbiota Modulation of Nutrient Properties in Chronic Kidney Disease.Adipose tissue immune response: novel triggers and consequences for chronic inflammatory conditions.Regulation of tissue factor gene expression in monocytes and endothelial cells: Thromboxane A2 as a new playerThe uremic toxicity of indoxyl sulfate and p-cresyl sulfate: a systematic review.Indoxyl sulfate downregulates expression of Mas receptor via OAT3/AhR/Stat3 pathway in proximal tubular cellsFunctional genomic analysis identifies indoxyl sulfate as a major, poorly dialyzable uremic toxin in end-stage renal disease.The cardiovascular effect of the uremic solute indole-3 acetic acid.Indoxyl sulfate-induced activation of (pro)renin receptor promotes cell proliferation and tissue factor expression in vascular smooth muscle cells.Thrombosis in the uremic milieu--emerging role of "thrombolome".Uraemic toxins and new methods to control their accumulation: game changers for the concept of dialysis adequacyModerate Increase of Indoxyl Sulfate Promotes Monocyte Transition into Profibrotic MacrophagesIndoxyl sulfate - the uremic toxin linking hemostatic system disturbances with the prevalence of cardiovascular disease in patients with chronic kidney disease.Indolic uremic solutes enhance procoagulant activity of red blood cells through phosphatidylserine exposure and microparticle releaseThe Aryl Hydrocarbon Receptor is a Critical Regulator of Tissue Factor Stability and an Antithrombotic Target in Uremia.Oxidized plasma albumin promotes platelet-endothelial crosstalk and endothelial tissue factor expression.Serum Indoxyl Sulfate Associates with Postangioplasty Thrombosis of Dialysis Grafts.Protein-bound uremic toxins stimulate crosstalk between leukocytes and vessel wall.Impact of Indoxyl Sulfate on Progenitor Cell-Related Neovascularization of Peripheral Arterial Disease and Post-Angioplasty Thrombosis of Dialysis Vascular Access.Protein-bound uremic toxins: new culprits of cardiovascular events in chronic kidney disease patients.New methods and technologies for measuring uremic toxins and quantifying dialysis adequacy.The intestine and the kidneys: a bad marriage can be hazardous.Future research directions to improve fistula maturation and reduce access failure.The Uremic Toxin Indoxyl Sulfate Accelerates Thrombotic Response after Vascular Injury in Animal Models.Indole 3-acetic acid, indoxyl sulfate and paracresyl-sulfate do not influence anemia parameters in hemodialysis patients.Inhibition of VEGF-Induced VEGFR-2 Activation and HUVEC Migration by Melatonin and Other Bioactive Indolic Compounds.The rebirth of interest in renal tubular function.Indoxyl sulfate suppresses hepatic fetuin-A expression via the aryl hydrocarbon receptor in HepG2 cells.Auxin induces cell proliferation in an experimental model of mammalian renal tubular epithelial cells.Indoxyl Sulfate: A Novel Cardiovascular Risk Factor in Chronic Kidney Disease.Vasculopathy in the setting of cardiorenal syndrome: roles of protein-bound uremic toxins.The role of adipose tissue asymmetric dimethylarginine/dimethylarginine dimethylaminohydrolase pathway in adipose tissue phenotype and metabolic abnormalities in subtotally nephrectomized rats.Dialysis Vascular Access Intervention and the Search for Biomarkers.Microbiome perturbation by oral vancomycin reduces plasma concentration of two gut-derived uremic solutes, indoxyl sulfate and p-cresyl sulfate, in end-stage renal disease.Indole-3-propionic acid suppresses indoxyl sulfate-induced expression of fibrotic and inflammatory genes in proximal tubular cells.Biochemical and Clinical Impact of Organic Uremic Retention Solutes: A Comprehensive Update.Targeting STUB1-tissue factor axis normalizes hyperthrombotic uremic phenotype without increasing bleeding risk.From bench to the hemodialysis clinic: protein-bound uremic toxins modulate NF-κB/Nrf2 expression.
P2860
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P2860
Indolic uremic solutes increase tissue factor production in endothelial cells by the aryl hydrocarbon receptor pathway.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
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name
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@en
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@nl
type
label
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@en
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@nl
prefLabel
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@en
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@nl
P2093
P356
P1433
P1476
Indolic uremic solutes increas ...... hydrocarbon receptor pathway.
@en
P2093
Anneleen Pletinck
Ariane Duval-Sabatier
Bertrand Gondouin
Claire Cerini
Françoise Dignat-George
Laetitia Dou
Laurent Arnaud
Marion Sallée
Noémie Jourde-Chiche
Philippe Brunet
P2888
P304
P356
10.1038/KI.2013.133
P407
P577
2013-05-01T00:00:00Z