Evidence for the nitric oxide pathway as a potential mode of action in fenoldopam-induced vascular injury.
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Evaluation of von Willebrand factor and von Willebrand factor propeptide in models of vascular endothelial cell activation, perturbation, and/or injuryProceedings of the 2014 National Toxicology Program Satellite SymposiumCaveolin-1 regulates the expression of tight junction proteins during hyperoxia-induced pulmonary epithelial barrier breakdown.Nonclinical safety biomarkers of drug-induced vascular injury: current status and blueprint for the future.Tissue-engineered blood vessels as promising tools for testing drug toxicity.Sampling the Rat Mesenteric Artery.The relationship of glucokinase activator-induced hypoglycemia with arteriopathy, neuronal necrosis, and peripheral neuropathy in nonclinical studies.The role of eNOS phosphorylation in causing drug-induced vascular injury.
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P2860
Evidence for the nitric oxide pathway as a potential mode of action in fenoldopam-induced vascular injury.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@en
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@nl
type
label
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@en
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@nl
prefLabel
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@en
Evidence for the nitric oxide ...... dopam-induced vascular injury.
@nl
P2860
P356
P1476
Evidence for the nitric oxide ...... ldopam-induced vascular injury
@en
P2093
Calvert S Louden
David A Brott
P2860
P304
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10.1177/0192623312444027
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P577
2012-05-01T00:00:00Z