Cytokine-mediated inactivation of malarial gametocytes is dependent on the presence of white blood cells and involves reactive nitrogen intermediates.
about
A malaria transmission-directed model of mosquito life cycle and ecologyAmbivalent Outcomes of Cell Apoptosis: A Barrier or Blessing in Malaria ProgressionThe cell biology of malaria infection of mosquito: advances and opportunitiesMalaria Parasites: The Great EscapeParasite killing in malaria non-vector mosquito Anopheles culicifacies species B: implication of nitric oxide synthase upregulationEcological influences on the behaviour and fertility of malaria parasitesCationic amino acid transporters play key roles in the survival and transmission of apicomplexan parasitesProtein S-nitrosylation in Plasmodium falciparum.Mapping of specific and promiscuous HLA-DR-restricted T-cell epitopes on the Plasmodium falciparum 27-kilodalton sexual stage-specific antigen.Induction of nitric oxide synthase in Anopheles stephensi by Plasmodium falciparum: mechanism of signaling and the role of parasite glycosylphosphatidylinositols.Sex and death: the effects of innate immune factors on the sexual reproduction of malaria parasites.Phagocytosis does not play a major role in naturally acquired transmission-blocking immunity to Plasmodium falciparum malariaMalaria-induced changes in host odors enhance mosquito attraction.Naturally occurring triggers that induce apoptosis-like programmed cell death in Plasmodium berghei ookinetes.The Plasmodium bottleneck: malaria parasite losses in the mosquito vectorEpidemiology and infectivity of Plasmodium falciparum and Plasmodium vivax gametocytes in relation to malaria control and eliminationP. falciparum infection durations and infectiousness are shaped by antigenic variation and innate and adaptive host immunity in a mathematical model.Mammalian transforming growth factor beta1 activated after ingestion by Anopheles stephensi modulates mosquito immunity.Parasite virulence and disease patterns in Plasmodium falciparum malaria.Leukocytes in a Plasmodium falciparum-infected blood meal reduce transmission of malaria to Anopheles mosquitoes.Consequences of immunopathology for pathogen virulence evolution and public health: malaria as a case studyThe mosquito Anopheles stephensi limits malaria parasite development with inducible synthesis of nitric oxide.Low levels of mammalian TGF-beta1 are protective against malaria parasite infection, a paradox clarified in the mosquito hostMalaria parasite diversity and transmission intensity affect development of parasitological immunity in a mathematical model.Malaria transmission blocking immunity and sexual stage vaccines for interrupting malaria transmission in Latin America.A mathematical model of the impact of present and future malaria vaccinesHost immune constraints on malaria transmission: insights from population biology of within-host parasites.In vivo, in vitro, and in silico studies suggest a conserved immune module that regulates malaria parasite transmission from mammals to mosquitoesHuman host-derived cytokines associated with Plasmodium vivax transmission from acute malaria patients to Anopheles darlingi mosquitoes in the Peruvian AmazonInformation use and plasticity in the reproductive decisions of malaria parasites.Sera from patients with falciparum malaria induce substance P gene expression in cultured human brain microvascular endothelial cells.Modulation of transmission success of Plasmodium falciparum gametocytes (sexual stages) in various species of Anopheles by erythrocytic asexual stage parasites.Mechanisms underlying the monocyte-mediated antibody-dependent killing of Plasmodium falciparum asexual blood stagesNitric oxide production and nitric oxide synthase type 2 expression by human mononuclear phagocytes: a review.Host-parasite interactions revealed by Plasmodium falciparum metabolomics.Unravelling the immune signature of Plasmodium falciparum transmission-reducing immunity.Infection of Anopheles aquasalis from symptomatic and asymptomatic Plasmodium vivax infections in Manaus, western Brazilian Amazon.
P2860
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P2860
Cytokine-mediated inactivation of malarial gametocytes is dependent on the presence of white blood cells and involves reactive nitrogen intermediates.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh-hant
name
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@en
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@nl
type
label
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@en
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@nl
prefLabel
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@en
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@nl
P2860
P1433
P1476
Cytokine-mediated inactivation ...... active nitrogen intermediates.
@en
P2093
Karunaweera ND
Naotunne TS
P2860
P304
P577
1993-04-01T00:00:00Z