Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
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NFATc3 mediates chronic hypoxia-induced pulmonary arterial remodeling with alpha-actin up-regulationEndothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries.Dehydroepiandrosterone upregulates soluble guanylate cyclase and inhibits hypoxic pulmonary hypertension.The principal pathways involved in the in vivo modulation of hypoxic pulmonary vasoconstriction, pulmonary arterial remodelling and pulmonary hypertension.Chronic hypoxia alters fetal cerebrovascular responses to endothelin-1.Altered Redox Balance in the Development of Chronic Hypoxia-induced Pulmonary Hypertension.Determination of darusentan enantiomers in rat plasma by enantioselective liquid chromatography with tandem mass spectrometry using cellulose-based chiral stationary phase
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P2860
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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name
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@en
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@nl
type
label
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@en
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@nl
prefLabel
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@en
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@nl
P2093
P2860
P1476
Effects of ET-A receptor blockade on eNOS gene expression in chronic hypoxic rat lungs.
@en
P2093
Cornelia Lorenz
Friedrich C Blumberg
Günter A J Riegger
Konrad Wolf
Michael Arzt
Michael Pfeifer
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P304
P356
10.1152/JAPPLPHYSIOL.00239.2002
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P577
2002-10-04T00:00:00Z